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Dual Innovations in EGFR Degradation and Antigen-Specific Modulation for Multiple Sclerosis
Anna C Renner1, Robert B Kargbo2
1North Dakota State University, Fargo, North Dakota 58108-6050, United States.
None:
Recent inventions have unveiled two complementary therapeutic strategies: cyclic peptides that trigger the lysosomal degradation of the epidermal growth factor receptor (EGFR), and soluble peptide-MHC constructs that suppress autoreactive T-cell receptors in multiple sclerosis. Together, these innovations highlight emerging frameworks for precision control of receptor signaling and adaptive immunity using structurally defined peptide modulators.
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