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Crystal Field Theory - Octahedral Complexes

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A New Approach for the Comparative Analysis of Multiprotein Complexes Based on 15N Metabolic Labeling and Quantitative Mass Spectrometry

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Assessing Evidence to Include Filifactor alocis as a Novel Candidate in Socransky's Complexes.

Ali A Abdulkareem¹, Sarhang S Gul^2,3, Hayder R Abdulbaqi¹

¹Department of Periodontics, College of Dentistry, University of Baghdad, Baghdad, Iraq.

Molecular Oral Microbiology

|January 15, 2026

Summary

Filifactor alocis is a potential periodontal pathogen. While not yet sufficient for Socransky

Keywords:

Filifactor alocis Socransky's complexes high‐throughput nucleotide sequencing metagenomics periodontal diseases periodontitis

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Area of Science:

Microbiology and Periodontology

Background:

Socransky's complexes historically define key bacteria in periodontal disease.
Advancements in microbiome analysis allow detailed investigation of periodontal pathogens.
Filifactor alocis has recently gained attention as a potential periodontal pathogen.

Purpose of the Study:

To review existing evidence on Filifactor alocis's role in periodontal disease.
To assess the potential for including Filifactor alocis in Socransky's periodontal disease model.

Main Methods:

Comprehensive literature search of PubMed, ScienceDirect, and Scopus databases.
Inclusion of 24 observational, clinical, in vitro, and animal studies.
Synthesis of data on prevalence, host response, and virulence factors.

Main Results:

Higher prevalence of F. alocis observed at diseased periodontal sites compared to healthy sites.
Periodontal treatment significantly reduced F. alocis levels and improved clinical parameters.
In vitro and experimental studies indicate F. alocis possesses virulence attributes similar to known periodontal pathogens.

Conclusions:

Current evidence is insufficient to formally include F. alocis in Socransky's complexes.
F. alocis shows potential as a future addition to Socransky's model.
Further highly standardized research is required to confirm F. alocis's pathogenic role and comparability.