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Related Concept Videos

DNA as a Genetic Template02:05

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Two structural features of the DNA molecule provide a basis for the mechanisms of heredity: the four nucleotide bases and its double-stranded nature. The Watson-Crick model of double-helical DNA structure, proposed in 1952, drew heavily upon the X-ray crystallography work of researchers Rosalind Franklin and Maurice Wilkins. Watson, Crick, and Wilkins jointly received the Nobel Prize in Physiology or Medicine for their work in 1962. Franklin was, controversially, excluded from the prize for...
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The genomes of eukaryotes are punctuated by long stretches of sequence which do not code for proteins or RNAs. Although some of these regions do contain crucial regulatory sequences, the vast majority of this DNA serves no known function. Typically, these regions of the genome are the ones in which the fastest change, in evolutionary terms, is observed, because there is typically little to no selection pressure acting on these regions to preserve their sequences.
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The Evidence for Evolution02:55

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Genetic variations accumulating within populations over generations give rise to biological evolution. Evolutionary changes can result in the formation of novel varieties and entire new species. These changes are responsible for the diverse forms of life inhabiting the planet. The evidence for evolution suggests that all living organisms descended from common ancestors.
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Updated: Mar 22, 2026

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DNA as a quantum system in evolution.

Nahuel Aquiles Garcia1

  • 1GECORP. Av. Juan Manuel de Rosas 899. S. M. del Monte, Buenos Aires, Argentina.

Plos One
|March 20, 2026
PubMed
Summary

Time emerges from increasing information entropy, influencing DNA replication and repair via quantum effects. Weak perturbations subtly alter mutation probabilities, linking cellular processes to cosmic time and suggesting new experiments.

Area of Science:

  • Quantum Biology
  • Information Theory
  • Genomics

Background:

  • Time's emergence is explored through information entropy.
  • DNA is modeled as an open quantum system influenced by perturbations.
  • Micro-timing shifts in DNA replication/repair can alter mutation probabilities.

Purpose of the Study:

  • To investigate a quantum-information model where time emerges from increasing information entropy.
  • To explore how weak, time-dependent perturbations affect DNA replication, repair, and mutation probabilities.
  • To connect cellular aging and evolution to cosmic time via DNA dynamics.

Main Methods:

  • A toy quantum-information model of DNA as an open quantum system.
  • Mapping Mycobacterium tuberculosis genome nucleotides to qubit states.

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  • Quantifying informational structure using Shannon and von Neumann entropies and coding/non-coding correlations.
  • Simulating Hamiltonian dynamics under physiological perturbations and external signals.

    • Main Results:

    • Genomic segments exhibit distinctive dynamical signatures under plausible perturbations compared to controls.
    • Weak perturbations bias micro-timing, influencing tautomeric states and mutation probabilities.
    • Sequence-dependent responses to perturbations generate testable predictions for mutation spectra shifts.

    Conclusions:

    • The study proposes a framework connecting cellular aging and evolution to cosmic time.
    • DNA's sensitivity to time-dependent perturbations is highlighted.
    • The model suggests experiments to probe these quantum effects on mutation patterns.