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Related Concept Videos

Rheumatic Heart Disease III: Medical Management01:21

Rheumatic Heart Disease III: Medical Management

Rheumatic heart disease (RHD) management can be divided into two main strategies: prevention and long-term management.Primary PreventionPrimary prevention focuses on timely diagnosis and management of group A streptococcal pharyngitis to prevent acute rheumatic fever. The most widely used antibiotic for treating this condition is intramuscular benzathine penicillin G.Acute Rheumatic Fever TreatmentThe primary treatment goal for a patient diagnosed with acute rheumatic fever is to suppress the...
Rheumatic Heart Disease I: Introduction01:23

Rheumatic Heart Disease I: Introduction

Rheumatic heart disease or RHD is a chronic condition that results from rheumatic fever, causing permanent damage to the heart valves.Etiology and Risk FactorsIt primarily arises from rheumatic fever, an inflammatory disease that can develop after untreated or inadequately treated group A streptococcal (GAS) pharyngitis. Streptococcus spreads through direct contact with oral or respiratory secretions. While the bacteria are the causative agents, factors like malnutrition, overcrowding, poor...
Rheumatic Heart Disease II: Clinical Manifestations and Diagnostic Studies01:22

Rheumatic Heart Disease II: Clinical Manifestations and Diagnostic Studies

The key clinical manifestations of Rheumatic heart disease (RHD) include several distinct cardiac symptoms.Carditis, a hallmark of acute rheumatic fever, involves inflammation of the heart's endocardium, myocardium, and pericardium. Chronic RHD often results from recurrent episodes of carditis. Its symptoms include the following:Murmurs are caused by valvular damage, especially to the mitral and aortic valves. Mitral stenosis or regurgitation is common, with characteristic heart murmurs...

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Updated: May 19, 2026

Transradial Access Chemoembolization for Hepatocellular Carcinoma Patients
05:31

Transradial Access Chemoembolization for Hepatocellular Carcinoma Patients

Published on: September 20, 2020

Rheumatoid arthritis: transarterial microembolization for refractory localized joint inflammation.

You-Chien Lin1, Jia-Min Wu2,3, Cheng-Chun Lee4

  • 1Division of Diagnostic Radiology, Department of Medical Imaging, Tungs' Taichung Metroharbor Hospital, No.699, Section 8, Taiwan Boulevard, Wuqi District, Taichung City, 43503, Taiwan (R.O.C.).

Skeletal Radiology
|May 18, 2026
PubMed
Summary
This summary is machine-generated.

Transarterial microembolization (TAME) offers a localized treatment for persistent joint pain in rheumatoid arthritis. This minimally invasive procedure reduced neovascularization, improving pain and function in a patient case study.

Keywords:
MicroembolizationNeovascularizationTransarterial microembolization (TAME)

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Published on: May 16, 2025

Area of Science:

  • Interventional Radiology
  • Rheumatology
  • Vascular Medicine

Background:

  • Rheumatoid arthritis (RA) can cause persistent localized joint pain despite systemic therapy.
  • Periarticular neovascularization contributes to inflammation and pain in RA.
  • Current systemic treatments may not fully address localized inflammatory processes.

Purpose of the Study:

  • To evaluate the safety and efficacy of transarterial microembolization (TAME) as a localized treatment for RA.
  • To assess TAME's impact on periarticular neovascularization and associated symptoms.
  • To explore TAME as an adjunct to existing systemic immunomodulatory therapy for RA.

Main Methods:

  • Selective transarterial microembolization targeting angiographically identified neovessels in affected joints.
  • Intra-arterial delivery of imipenem/cilastatin during the procedure.
  • Assessment of hypervascularity via post-embolization angiography.
  • Clinical evaluation of pain and function over 12 months.
  • Magnetic resonance imaging (MRI) to assess changes in synovial proliferation, edema, and bone marrow edema.

Main Results:

  • Post-embolization angiography showed reduced hypervascularity.
  • Significant improvement in pain and joint function over a 12-month follow-up period.
  • MRI revealed decreased synovial proliferation, periarticular edema, and bone marrow edema.
  • The patient's systemic therapy regimen remained unchanged.
  • Adverse events were transient, including procedure-related pain and skin discoloration.

Conclusions:

  • Transarterial microembolization (TAME) demonstrates potential as a localized adjunct therapy for rheumatoid arthritis.
  • TAME effectively reduced neovascularization and improved clinical outcomes in a case study.
  • Further prospective studies are warranted to validate TAME for RA management.