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Updated: May 23, 2026

Modeling Encephalopathy of Prematurity Using Prenatal Hypoxia-ischemia with Intra-amniotic Lipopolysaccharide in Rats
07:36

Modeling Encephalopathy of Prematurity Using Prenatal Hypoxia-ischemia with Intra-amniotic Lipopolysaccharide in Rats

Published on: November 20, 2015

Preterm Birth and Maternal Cardiovascular Risk.

Jorge Segovia-Reyes1,2, Andrea Iboleon-Jimenez3, María Jiménez-Salva1,2

  • 1Cardiology and Cardiovascular Surgery Department, Hospital Universitario Virgen de la Victoria Málaga, Spain.

European Cardiology
|May 22, 2026
PubMed
Summary
This summary is machine-generated.

Preterm birth (PTB) is a significant predictor of future cardiovascular disease, including hypertension and heart disease. Recognizing PTB as a risk marker improves cardiovascular risk assessment and prevention strategies for women.

Keywords:
Preterm birthcardiovascular diseaseendothelial dysfunctioninflammationmaternal cardiovascular risk

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A Rat Model of Mild Intrauterine Hypoperfusion with Microcoil Stenosis
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Published on: January 7, 2018

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Last Updated: May 23, 2026

Modeling Encephalopathy of Prematurity Using Prenatal Hypoxia-ischemia with Intra-amniotic Lipopolysaccharide in Rats
07:36

Modeling Encephalopathy of Prematurity Using Prenatal Hypoxia-ischemia with Intra-amniotic Lipopolysaccharide in Rats

Published on: November 20, 2015

A Rat Model of Mild Intrauterine Hypoperfusion with Microcoil Stenosis
06:19

A Rat Model of Mild Intrauterine Hypoperfusion with Microcoil Stenosis

Published on: January 7, 2018

Area of Science:

  • Cardiology
  • Obstetrics
  • Epidemiology

Background:

  • Preterm birth (PTB) is linked to immediate perinatal risks.
  • Growing evidence indicates PTB predicts long-term cardiovascular disease (CVD).
  • Women with PTB history show higher risks for hypertension, diabetes, coronary heart disease, stroke, and CVD mortality.

Purpose of the Study:

  • To summarize epidemiological evidence linking PTB to future cardiovascular risk.
  • To explore proposed biological mechanisms (inflammation, endothelial dysfunction, oxidative stress, placental abnormalities, genetic/epigenetic factors).
  • To discuss clinical implications and provide recommendations.

Main Methods:

  • Review of epidemiological evidence.
  • Exploration of biological mechanisms.
  • Discussion of clinical implications and guidelines.

Main Results:

  • PTB is an independent cardiovascular risk marker.
  • Significant associations found between PTB history and long-term CVD risks.
  • Biological mechanisms involve inflammation, endothelial dysfunction, oxidative stress, and placental issues.

Conclusions:

  • Integrating obstetric history into cardiovascular risk assessment is crucial.
  • Recognizing PTB improves cardiovascular risk stratification and prevention.
  • Routine care incorporating PTB history can enhance long-term cardiovascular outcomes for women.