Related Experiment Video
Updated: Jul 9, 2026

Open Tracheostomy Gastric Acid Aspiration Murine Model of Acute Lung Injury Results in Maximal Acute Nonlethal Lung Injury
Published on: February 26, 2017
Acute oesophageal necrosis following ethyl-induced emesis: A Case Report
V Van Lint1, S Van Gool1, G Van Roey1
1Department of Gastro-enterology, AZ Turnhout, Turnhout, Belgium.
Abstract:
Acute oesophageal necrosis, also known as black oesophagus or Gurvits syndrome, is a rare condition characterized by black discoloration of the distal oesophageal mucosa due to mucosal necrosis, typically with an abrupt transition at the gastroesophageal junction. We report a case of Gurvits syndrome in a 73-year-old man with poorly controlled type 2 diabetes mellitus and recent alcohol-induced vomiting, without ingestion of caustic substances. The patient experienced epigastric pain while remaining hemodynamically stable without significant biochemical abnormalities. Following supportive therapy with bowel rest, intravenous fluids, acid suppression, glycemic control and empiric antibiotics followed by enteral feeding via jejunostomy, patient recovered completely without complications. We observed progressive mucosal healing on follow-up endoscopy. This case highlights the importance of early recognition and supportive management of acute oesophageal necrosis, especially in patients with predisposing factors such as diabetes mellitus, vomiting, to achieve full recovery and avoid adverse outcomes.
Related Concept Videos
Esophageal Perforation-II: Clinical Manifestations and Management
Clinical Manifestations:
Esophageal Perforation-I: Introduction
The location of esophageal perforation can vary, occurring anywhere along the esophagus.
Esophageal Achalasia
Esophageal Strictures-I: Introduction
Etiology
The primary cause of esophageal strictures is long-standing gastroesophageal reflux disease (GERD), accounting for about 70 to 80% of adult cases. Chronic acid reflux can lead to injury and scarring of the esophageal lining, culminating in...
Gastroesophageal Reflux Disease
Intestinal Obstruction II: Pathophysiology
