Related Experiment Video
Updated: Aug 5, 2026
02:30
Endoscopic Approach for Colloid Cyst Resection
Published on: May 23, 2025
Clinical and Radiological Features of a Large Intracranial Hydatid Cyst: A New Case Report
Kazem Ghaemi1, Mahmoodreza Behravan2, Soudabe Eshaghi2
1Department of Neurosurgery, Birjand University Faculty of Medicine, Birjand, Iran.
Case Reports in Infectious Diseases
|July 31, 2026
Summary
Intracranial hydatid cysts, caused by Echinococcus granulosus, are rare but serious. This case highlights the successful surgical removal and albendazole treatment of a temporal lobe hydatid cyst in a child.
Area of Science:
- Neurology
- Parasitology
- Infectious Diseases
Background:
- Intracranial hydatid cysts are rare, potentially fatal parasitic infections.
- Caused by the larval stage of Echinococcus granulosus.
- Often present with non-specific neurological symptoms.
Purpose of the Study:
- To report a case of intracranial hydatid cyst in a pediatric patient.
- To describe the diagnostic and surgical management.
- To emphasize the importance of considering hydatid cysts in differential diagnosis.
Main Methods:
- Case report of an 11-year-old boy with neurological symptoms.
- Neuroimaging (MRI/CT) to identify the cystic lesion.
- Surgical excision using the Dowling technique.
- Postoperative albendazole therapy.
Main Results:
- A large left temporal lobe cystic lesion with mass effect was identified.
- Surgical removal was successful without rupture.
- The patient experienced a good recovery with no neurological deficits.
- Histopathological confirmation of Echinococcus granulosus.
Conclusions:
- Intracranial hydatid cysts should be considered in the differential diagnosis of brain cystic lesions.
- Early diagnosis and appropriate surgical intervention are crucial.
- Albendazole is an important adjunct therapy for hydatid disease.
Related Concept Videos
01:26Brain Abscess l: Introduction
A brain abscess is a focal, intracerebral infection characterized by a localized collection of pus within the brain parenchyma, resulting from microbial invasion and the body’s inflammatory response. It progresses through stages: early and late cerebritis, followed by early and late capsule formation, reflecting tissue destruction, immune response, and eventual encapsulation.Etiology and PathogenesisCausative organisms vary with source and host factors, often involving polymicrobial infections,...
01:14Increased Intracranial Pressure l: Introduction
Intracranial hypertension is a sustained elevation of intracranial pressure (ICP) above 22 mm Hg. In supine adults, normal ICP is ~7–15 mm Hg.The rigid, nonexpandable cranium contains three components—brain tissue, blood, and cerebrospinal fluid (CSF)—that total ~1,700 mL in a typical adult: 1,400 mL brain (~80%), 150 mL blood (~10%), and 150 mL CSF (~10%). According to the Monro–Kellie doctrine, total intracranial volume is effectively fixed. When one component expands, CSF and venous blood...
01:28Amebiasis
Entamoeba histolytica, a protozoan parasite, is responsible for intestinal and extraintestinal amebiasis. Though a significant proportion of infections remain asymptomatic, approximately 50 million individuals annually are estimated to present with clinical disease, resulting in up to 100,000 deaths globally. The disease burden is disproportionately high in regions with lower socioeconomic status, such as parts of India, Africa, Mexico, and Latin America.Etiology and TransmissionThe infective...
01:29Increased Intracranial Pressure ll: Pathophysiology
Increased intracranial pressure (ICP) refers to a potentially life-threatening rise in pressure inside the skull. This usually happens when there is a major change in the volume of brain tissue, blood, or cerebrospinal fluid (CSF) — the three components inside the skull. According to the Monro-Kellie doctrine, if the volume of one component increases, the volumes of the other components must decrease to maintain normal pressure. If this does not happen, ICP rises.The process often begins with...