Do ultraviolet-induced mutations to streptomycin resistance exhibit susceptibility to mutation frequency decline?

Mutation frequency decline revisited.

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An SOS-inducible defective retronphage (phi R86) in Escherichia coli strain B.

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Overproduction of DnaE protein (alpha subunit of DNA polymerase III) restores viability in a conditionally inviable Escherichia coli strain deficient in DNA polymerase I.

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Escherichia coli mfd mutant deficient in "mutation frequency decline" lacks strand-specific repair: in vitro complementation with purified coupling factor.

Proceedings of the National Academy of Sciences of the United States of America·1991

Novel SOS phenotypes caused by second-site mutations in the recA430 gene of Escherichia coli.

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RecA protein in the SOS response: milestones and mysteries.

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In silico analysis of missense variants in functional domains of PWWP2A and PWWP2B proteins.

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The role of KIT mutations in oral melanoma and disease progression: A literature review on early detection and targeted therapy.

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Non-clonal interphase markers of chromosomal instability in malignancy acquisition and cancer progression.

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miR-145-5p induces ferroptosis by targeting Notch2 to suppress melanoma progression.

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The evolutionary impact of Robertsonian fusions: linking genomic architecture, recombination and reproduction.

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How Did a Size-Divergent Water Strider Species Group Exhibiting an East Asia-Eastern North America Disjunction Originate and Diversify?

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Gene Genealogies in Haploid Populations Evolving According to Sweepstakes Reproduction.

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Emerging epidemiology of invasive group a Streptococcus in tropical Australia: a 21-year population-based data linkage study.

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Related Experiment Videos

Do ultraviolet-induced mutations to streptomycin resistance exhibit susceptibility to mutation frequency decline?

Related Concept Videos

Mutation frequency decline revisited.

An SOS-inducible defective retronphage (phi R86) in Escherichia coli strain B.

Overproduction of DnaE protein (alpha subunit of DNA polymerase III) restores viability in a conditionally inviable Escherichia coli strain deficient in DNA polymerase I.

Escherichia coli mfd mutant deficient in "mutation frequency decline" lacks strand-specific repair: in vitro complementation with purified coupling factor.

Novel SOS phenotypes caused by second-site mutations in the recA430 gene of Escherichia coli.

RecA protein in the SOS response: milestones and mysteries.

In silico analysis of missense variants in functional domains of PWWP2A and PWWP2B proteins.

The role of KIT mutations in oral melanoma and disease progression: A literature review on early detection and targeted therapy.

Mothers against decapentaplegic homolog 3-activated FAT atypical cadherin 2 regulates programmed death ligand-1 to mediate immune evasion in lung squamous cell carcinoma.

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Non-clonal interphase markers of chromosomal instability in malignancy acquisition and cancer progression.

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The evolutionary impact of Robertsonian fusions: linking genomic architecture, recombination and reproduction.

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Do ultraviolet-induced mutations to streptomycin resistance exhibit susceptibility to mutation frequency decline?

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