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Left ventricular size following endurance, sprint, and strength training
Medicine and Science in Sports and Exercise
|January 1, 1982
Summary
Endurance, sprint, and strength training improved aerobic capacity and strength but did not significantly alter left ventricular dimensions in adolescent boys. Small increases in calculated left ventricular mass were likely due to bradycardia, not hypertrophy.
Area of Science:
- Exercise Physiology
- Cardiovascular Adaptations
- Sports Science
Background:
- Understanding cardiac adaptations to different training types is crucial for optimizing athletic performance and cardiovascular health.
- Adolescent athletes may exhibit distinct responses to training compared to adults.
Purpose of the Study:
- To investigate the effects of endurance, sprint, and strength training on left ventricular dimensions in adolescent boys.
- To determine if specific training adaptations lead to changes in cardiac structure.
Main Methods:
- Adolescent boys underwent three distinct training regimens: endurance (END), sprint (SPR), and strength (STR).
- Left ventricular dimensions, including interventricular and posterior wall thickness and internal diameter, were measured before and after training.
- Aerobic capacity (VO2max) and upper body strength were assessed to confirm training adaptations.
Main Results:
- Endurance and sprint training significantly increased VO2max.
- Strength training significantly improved upper body strength.
- No significant changes were observed in interventricular or left ventricular posterior wall thickness, or left ventricular internal diameter across all groups.
- A slight, significant increase in calculated left ventricular mass was noted in the END (10%) and STR (4%) groups.
Conclusions:
- Significant improvements in aerobic capacity and strength can be achieved without substantial modifications in left ventricular dimensions.
- The minor increases in calculated left ventricular mass are likely attributed to physiological responses like training bradycardia and increased diastolic filling time, rather than true cardiac hypertrophy.