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Related Experiment Videos

[Herpes zoster].

P Fritsch

    Wiener Klinische Wochenschrift
    |December 25, 1981
    PubMed
    Summary
    This summary is machine-generated.

    Herpes zoster, or shingles, is caused by varicella-zoster virus (VZV) reactivation from sensory ganglia. Early corticosteroid and antiviral treatment can manage pain and complications.

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    Area of Science:

    • Virology
    • Immunology
    • Dermatology

    Context:

    • Herpes zoster (shingles) pathogenesis and therapeutic strategies.
    • Evolving understanding of varicella-zoster virus (VZV) latency and reactivation.
    • Clinical presentation and complications of herpes zoster.

    Purpose:

    • To review current knowledge on herpes zoster pathogenesis.
    • To outline effective therapeutic interventions for herpes zoster.
    • To discuss the role of corticosteroids and antiviral agents in managing shingles.

    Summary:

    • Herpes zoster pathogenesis involves VZV reactivation from dorsal root ganglia, not reinfection.
    • Systemic corticosteroids, particularly in older adults, can reduce postherpetic neuralgia frequency and severity.
  • Antiviral drugs like vidarabine and acyclovir offer effective treatment with good tolerability.

    • Impact:

    • Clarifies VZV pathogenesis, shifting from reinfection to reactivation model.
    • Provides evidence-based guidelines for early-stage herpes zoster management.
    • Highlights the benefits of corticosteroids and antivirals in reducing shingles complications.