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Related Experiment Videos

Neonatal septic arthritis.

M Dan

    Israel Journal of Medical Sciences
    |November 1, 1983
    PubMed
    Summary
    This summary is machine-generated.

    Neonatal septic arthritis (NSA) microbiology differs based on acquisition source. Hospital-acquired NSA is often staphylococcal, while community-acquired NSA is increasingly streptococcal.

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    Area of Science:

    • Pediatrics
    • Infectious Diseases
    • Microbiology

    Background:

    • Neonatal septic arthritis (NSA) is a serious joint infection in newborns.
    • Understanding the causative pathogens is crucial for effective treatment and prevention.

    Purpose of the Study:

    • To correlate the microbiology of NSA with its clinical presentation.
    • To differentiate NSA pathogens based on hospital-acquired versus community-acquired infections.

    Main Methods:

    • Retrospective review of nine infants diagnosed with NSA between 1964-1981.
    • Analysis of 92 additional NSA cases reported in English literature since 1960.

    Main Results:

    • Hospital-acquired NSA predominantly involved staphylococci (62%), followed by Candida species (17%) and gram-negative enteric bacilli (15%).

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  • Community-acquired NSA was most frequently caused by streptococci (52%), followed by staphylococci (26%) and gonococci (17%).
  • Since 1970, community-acquired NSA shows a marked shift from staphylococcal (5%) to streptococcal (75%) isolates.

    • Conclusions:

    • The microbiological profile of NSA is distinct between hospital-acquired and community-acquired cases.
    • A significant epidemiological shift towards streptococcal dominance in community-acquired NSA has occurred since 1970.
    • These findings highlight the importance of source-specific microbiological data for managing NSA.