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Related Experiment Videos

Secondary vascular glaucoma.

R J de Keizer

    Documenta Ophthalmologica. Advances in Ophthalmology
    |December 15, 1983
    PubMed
    Summary
    This summary is machine-generated.

    Secondary vascular glaucoma, caused by venous obstruction, can manifest with elevated intraocular pressure. This study investigated its causes, including orbital and venous issues, finding a 2% glaucoma rate in exophthalmos patients.

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    Area of Science:

    • Ophthalmology
    • Vascular Medicine

    Background:

    • Secondary vascular glaucoma involves elevated intraocular pressure (IOP) stemming from venous obstructions.
    • Key etiological factors include lesions affecting vortex and anterior ciliary veins, as well as orbital and cavernous sinus obstructions.

    Purpose of the Study:

    • To investigate the prevalence and characteristics of secondary vascular glaucoma.
    • To explore the relationship between various orbital conditions and elevated IOP.

    Main Methods:

    • Retrospective analysis of patients with exophthalmos, tenonitis/scleritis posterior, and orbital pseudotumor.
    • Examination of patients with secondary vascular glaucoma due to elevated episcleral venous pressure.

    Main Results:

    • Only 2% of exophthalmos patients exhibited glaucoma, similar to the general population.

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  • Elevated IOP was found in 30% of patients with tenonitis/scleritis posterior.
  • Six female patients over 40 with idiopathic elevated episcleral venous pressure presented with secondary vascular glaucoma; three had associated orbital neuro-ophthalmological lesions.

    • Conclusions:

    • Secondary vascular glaucoma is linked to specific venous and orbital pathologies.
    • Idiopathic elevated episcleral venous pressure is a significant cause in certain demographics.
    • Orbital neuro-ophthalmological findings may co-exist with secondary vascular glaucoma.