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[Exocrine function of the pancreas in children]

Voprosy Pitaniia
|March 1, 1984
PubMed

Insights

Children with constitutional-exogenous obesity showed increased trypsin and trypsin inhibitor activity. This suggests the pancreas retains compensatory function, highlighting the importance of dietetics for managing this condition.

Area of Science:

  • Biochemistry
  • Pediatrics
  • Endocrinology

Context:

  • Investigating pancreatic proteolytic function in children with constitutional-exogenous obesity.
  • Comparing enzyme activity in obese children versus normal controls.
  • Utilizing established methods for trypsin and trypsin inhibitor assays.

Purpose:

  • To assess the activity of trypsin and its inhibitor in the blood serum of children with constitutional-exogenous obesity.
  • To understand the compensatory mechanisms of the pancreas in pediatric obesity.
  • To inform therapeutic strategies, particularly dietetics, for managing childhood obesity.

Summary:

  • Thirty-three children, including 14 girls and 7 boys with constitutional-exogenous obesity and 12 normal children, were studied.
  • Trypsin activity and blood serum trypsin inhibitor levels were measured using modified Erlanger and Haverback methods.
  • Increased activation of both trypsin and its inhibitor was observed in children with obesity, correlating with obesity severity.

Impact:

  • Findings indicate preserved compensatory capabilities of the pancreas in children with constitutional-exogenous obesity.
  • Highlights the potential role of dietetics in normalizing pancreatic function and managing childhood obesity.
  • Suggests that monitoring trypsin inhibitor activity may be valuable in assessing pancreatic health in obese children.

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