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Acquired acrodermatitis enteropathica.

C G Burkhart

    Cutis
    |June 1, 1981
    PubMed
    Summary
    This summary is machine-generated.

    Acrodermatitis enteropathica, a zinc deficiency disorder, presents in infantile and acquired forms. Reduced serum alkaline phosphatase can indicate this condition, as shown in a case study of acquired acrodermatitis enteropathica.

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    Area of Science:

    • Dermatology
    • Nutritional Science
    • Biochemistry

    Background:

    • Acrodermatitis enteropathica is a genetic or acquired disorder.
    • Both forms stem from zinc deficiency.
    • Total parenteral nutrition can lead to acquired acrodermatitis enteropathica.

    Observation:

    • This study examines the acquired form of acrodermatitis enteropathica.
    • A case of acquired acrodermatitis enteropathica is presented.
    • The utility of serum alkaline phosphatase as a diagnostic marker is explored.

    Findings:

    • Reduced serum alkaline phosphatase levels are a marker for acrodermatitis enteropathica.
    • Zinc deficiency underlies both hereditary and acquired forms of the condition.
    • The presented case illustrates the acquired form of the disease.

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    Implications:

    • Early identification of zinc deficiency is crucial for managing acrodermatitis enteropathica.
    • Monitoring serum alkaline phosphatase may aid in diagnosing this condition.
    • Understanding the link between TPN and zinc deficiency can prevent acquired acrodermatitis enteropathica.