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Cyclooxygenase-independent effects of non-steroidal anti-inflammatory drugs on the neutrophil respiratory burst.

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A novel conformationally restricted protein kinase C inhibitor, Ro 31-8425, inhibits human neutrophil superoxide generation by soluble, particulate and post-receptor stimuli.

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The protein kinase C inhibitor, K252a, inhibits superoxide production in human neutrophils activated by both PIP2-dependent and -independent mechanisms.

Biochemical pharmacology·1991

The effect of new potent selective inhibitors of protein kinase C on the neutrophil respiratory burst.

Biochemical and biophysical research communications·1990

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Agents and actions·1994

Diamine oxidase in relation to diamine and polyamine metabolism.

Agents and actions·1994

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The modulation of lymphocyte activation by HE receptor agonists [proceedings]

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The effect of non-steroidal anti-inflammatory drugs on the accumulation and release of interleukin-1-like activity by peritoneal macrophages from the mouse.

Cyclooxygenase-independent effects of non-steroidal anti-inflammatory drugs on the neutrophil respiratory burst.

A novel conformationally restricted protein kinase C inhibitor, Ro 31-8425, inhibits human neutrophil superoxide generation by soluble, particulate and post-receptor stimuli.

The protein kinase C inhibitor, K252a, inhibits superoxide production in human neutrophils activated by both PIP2-dependent and -independent mechanisms.

The effect of new potent selective inhibitors of protein kinase C on the neutrophil respiratory burst.

The effect of putative protein kinase C inhibitors, K252a and staurosporine, on the human neutrophil respiratory burst activated by both receptor stimulation and post-receptor mechanisms.

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Cryptococcal and EBV Meningitis in a Postpartum Woman With HIV Despite ART: A Case Challenging the Limits of Immune Reconstitution.

High-Dimensional Spectral Flow Cytometry Panel for Immunophenotypic Characterization of Fixed Whole Blood Samples.

Acute myeloid leukemia with unusual mature lymphoid-like morphology in a subset of blasts.

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The modulation of lymphocyte activation by HE receptor agonists [proceedings]

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