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Plasmid-determined resistance to fosfomycin in Serratia marcescens

Insights

Multiple-antibiotic-resistant Serratia marcescens strains transferred resistance genes to Escherichia coli. These transfers involved plasmids carrying linked resistance patterns, highlighting potential for multidrug resistance spread in hospitals.

Area of Science:

  • Microbiology
  • Molecular Biology
  • Genetics

Background:

  • Hospital-acquired infections pose significant challenges due to multidrug-resistant (MDR) pathogens.
  • Serratia marcescens is an opportunistic pathogen frequently implicated in hospital-acquired infections.
  • Understanding the mechanisms of antibiotic resistance transfer is crucial for infection control.

Purpose of the Study:

  • To investigate the conjugative transfer of antibiotic resistance from clinical isolates of Serratia marcescens to Escherichia coli.
  • To characterize the plasmids mediating antibiotic resistance transfer.
  • To identify the patterns of linked antibiotic resistance genes transferred.

Main Methods:

  • Conjugation experiments were performed to transfer antibiotic resistance from Serratia marcescens to Escherichia coli.
  • Transconjugant strains were analyzed for antibiotic resistance profiles.
  • Plasmid DNA was isolated and characterized by size (megadaltons).
  • Plasmid transformation into E. coli was performed.

Main Results:

  • Two distinct patterns of transferable antibiotic resistance were identified in Serratia marcescens.
  • The first pattern included resistance to carbenicillin, streptomycin, and fosfomycin.
  • The second, more common pattern, conferred resistance to a broader spectrum of antibiotics: carbenicillin, streptomycin, kanamycin, gentamicin, tetracycline, chloramphenicol, sulfonamide, and fosfomycin.
  • These resistance patterns were mediated by single plasmids of 57 or 97 megadaltons.
  • Both plasmids were present in fosfomycin-resistant parental S. marcescens strains.
  • The 57-megadalton plasmid was successfully transformed into E. coli.

Conclusions:

  • Serratia marcescens clinical isolates possess plasmids capable of transferring multiple antibiotic resistances to Escherichia coli via conjugation.
  • The identified plasmids carry linked resistance genes, contributing to the spread of multidrug resistance.
  • The findings underscore the importance of monitoring antibiotic resistance mechanisms in hospital settings to prevent the dissemination of MDR strains.

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