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The paper reports an unusual pathogenetic mechanism of the cerebral circulation disorder in hypertensive crisis. Cerebellar infarction developed as a result of compression in great occipitae foramen of the inferior posterior cerebellar artery which was a complication of a severe hypertensive crisis with cerebral edema and a shift of stem structures into the great occipitae foramen. Of great importance in the parthogenesis of infarction was a rarely occuring variant of the course of the right inferior posterior cerebellar artery.
The paper reports an unusual pathogenetic mechanism of the cerebral circulation disorder in hypertensive crisis. Cerebellar infarction developed as a result of compression in great occipitae foramen of the inferior posterior cerebellar artery which was a complication of a severe hypertensive crisis with cerebral edema and a shift of stem structures into the great occipitae foramen. Of great importance in the parthogenesis of infarction was a rarely occuring variant of the course of the right inferior posterior cerebellar artery.