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Drosophila-host genetic control of susceptibility to Drosophila C virus

M Thomas-Orillard1, B Jeune, G Cusset

  • 1Université Pierre-et-Marie Curie, Paris, France.

Genetics
|August 1, 1995
PubMed

Insights

Genetic analysis reveals that partial tolerance to Drosophila C virus (DCV) in fruit flies (Drosophila melanogaster) is a dominant trait. This tolerance is linked to genes on chromosome III, not sex-linked or maternal factors.

Area of Science:

  • * Virology
  • * Genetics
  • * Entomology

Background:

  • * Drosophila C virus (DCV) is a natural pathogen of the fruit fly, Drosophila melanogaster.
  • * Natural populations of D. melanogaster exhibit varying degrees of susceptibility to DCV infections.
  • * Understanding host-pathogen interactions is crucial for studying viral evolution and host defense mechanisms.

Purpose of the Study:

  • * To investigate the genetic basis of differential susceptibility to DCV in natural populations of D. melanogaster.
  • * To determine the mode of inheritance for partial tolerance to DCV.
  • * To identify the chromosomal location of genes conferring DCV tolerance.

Main Methods:

  • * Intra-abdominal inoculation of 15 geographical strains of D. melanogaster with DCV.
  • * Crossbreeding experiments using isofemale lines from susceptible and partially tolerant strains.
  • * Segregation analysis of F1 and F2 progeny to map the genetic basis of tolerance.

Main Results:

  • * Natural D. melanogaster populations displayed varying susceptibility to DCV.
  • * Partial tolerance to DCV was observed in one strain and found to be a dominant trait.
  • * Genetic analysis indicated that tolerance is not sex-linked or maternally inherited, but segregates with chromosome III.

Conclusions:

  • * A specific gene or genes on chromosome III control the dominant partial tolerance to DCV in D. melanogaster.
  • * The findings provide insights into the genetic architecture of host resistance to viral pathogens.
  • * Two hypotheses are proposed to explain the mechanism of chromosome III gene action in DCV tolerance.

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