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B-lymphocyte subpopulations.

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The TNF-family cytokine TL1A promotes allergic immunopathology through group 2 innate lymphoid cells.

Mucosal immunology·2013

IL-1β strikingly enhances antigen-driven CD4 and CD8 T-cell responses.

Cold Spring Harbor symposia on quantitative biology·2013

Fibrosin, a novel fibrogenic cytokine, modulates expression of myofibroblasts.

Experimental and molecular pathology·2006

T-bet is rapidly induced by interferon-gamma in lymphoid and myeloid cells.

Proceedings of the National Academy of Sciences of the United States of America·2001

A spliceosome-independent eukaryote generated by complete intron removal.

Cell·2026

Recent advances in Alzheimer's disease: From molecular mechanisms to therapeutic strategies.

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Bridging omics and physiology to build multimodal clocks of human aging.

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Rewriting Duchenne muscular dystrophy therapy.

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Photosynthetic medicine: Engineering a plant-derived nanofoundry to power mammalian cells.

Cell·2026

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Cell·2026

Innate Immune Memory Responses in Organ Transplantation.

Arteriosclerosis, thrombosis, and vascular biology·2026

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10-HDA attenuates inflammatory responses in LPS-activated RAW264.7 cells <i>in vitro</i> via the NUR77/NF-κB pathways and improving mitochondrial functions.

Frontiers in pharmacology·2026

The IL-10-producing NKT10 subset plays a critical role in preventing graft-versus-host-disease.

Frontiers in immunology·2026

Life-threatening multiorgan immune-related toxicities complicated by sepsis after anti-PD-1 therapy with complete tumor regression: a case report and literature review.

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The multifaceted role of NF-κB signaling in psoriasis: from inflammatory amplification to epidermal remodeling.

Frontiers in immunology·2026

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Cytokine regulation of lung Th17 response to airway immunization using LPS adjuvant.

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The TNF-family cytokine TL1A promotes allergic immunopathology through group 2 innate lymphoid cells.

IL-1β strikingly enhances antigen-driven CD4 and CD8 T-cell responses.

Fibrosin, a novel fibrogenic cytokine, modulates expression of myofibroblasts.

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