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[Demyelinating polyradiculitis in neuro borreliosis]
I Corral1, G Sanchís, G García-Ribas
1Servicio de Neurología, Hospital Ramón y Cajal, Madrid.
Neurologia (Barcelona, Spain)
|February 1, 1995
Summary
Demyelinating neuropathy is a rare Lyme borreliosis complication. This case shows Borrelia burgdorferi can cause demyelinating polyradiculitis affecting nerve roots and plexus without peripheral nerve damage.
Area of Science:
- Neurology
- Infectious Diseases
- Neuroimmunology
Background:
- Demyelinating neuropathy is an uncommon manifestation of Lyme borreliosis.
- Lyme borreliosis is caused by the bacterium Borrelia burgdorferi.
- Neurophysiological studies are crucial for diagnosing neuropathies.
Observation:
- A case of meningoradiculitis due to Borrelia burgdorferi infection was analyzed.
- Neurophysiological analysis revealed demyelination primarily affecting nerve roots and plexus.
- No significant peripheral nerve involvement or distal axonal damage was detected.
Findings:
- Borrelia burgdorferi infection can lead to demyelinating polyradiculitis.
- The neurophysiological findings indicate localized demyelination in the nerve roots and plexus.
- Absence of peripheral nerve damage differentiates this presentation.
Implications:
- This case expands the understanding of neurological complications of Lyme disease.
- It highlights the potential for Borrelia burgdorferi to cause specific patterns of demyelination.
- Clinicians should consider Lyme disease in cases of unexplained radiculoplexus demyelination.