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Myoclonus associated with lorazepam therapy in very-low-birth-weight infants

D S Lee1, H A Wong, D C Knoppert

  • 1Department of Paediatrics, St. Joseph's Health Centre, Lawson Research Institute, University of Western Ontario, London, Canada.

Biology of the Neonate
|January 1, 1994
PubMed

Insights

Lorazepam is increasingly used for sedation in newborns, but may cause myoclonus in very-low-birth-weight infants. Caution is advised when administering injectable lorazepam to these vulnerable infants.

Area of Science:

  • Neonatal pharmacology
  • Pediatric neurology
  • Clinical toxicology

Background:

  • Lorazepam is frequently administered as a sedative to neonates and young infants.
  • Increasing use raises concerns about the drug's safety profile in this population.
  • Particular attention is needed for very-low-birth-weight (VLBW) infants (< 1,500 g).

Observation:

  • Three VLBW infants (< 1,500 g) developed myoclonus after intravenous lorazepam.
  • Myoclonus is characterized by sudden, involuntary muscle jerks.
  • This adverse event occurred in a vulnerable neonatal subgroup.

Findings:

  • Intravenous lorazepam administration was associated with myoclonus in VLBW infants.
  • The study highlights potential neurotoxic effects of lorazepam and its vehicle.
  • This suggests a specific risk in extremely preterm or low-birth-weight neonates.

Implications:

  • Injectable lorazepam should be used with significant caution in VLBW infants.
  • Further research into lorazepam's neurotoxicity in neonates is warranted.
  • Clinical practice guidelines may need revision regarding lorazepam use in VLBW infants.

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