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[Convulsive disorder in celiac disease]

O Cohen1, Y River, I Zelinger

  • 1Dept. of Neurology, Hadassah-University Hospital, Jerusalem.

Harefuah
|June 15, 1994
PubMed
Summary
This summary is machine-generated.

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Patients with celiac disease experiencing hypocalcemia are at high risk for seizures. Prompt correction of low calcium levels is crucial to prevent convulsions in these individuals.

Area of Science:

  • Neurology
  • Gastroenterology
  • Endocrinology

Background:

  • Celiac disease is an autoimmune disorder affecting the small intestine.
  • Malabsorption in celiac disease can lead to deficiencies in essential minerals like calcium.
  • Neurological complications, including seizures, have been anecdotally linked to celiac disease.

Observation:

  • A 40-year-old woman with diagnosed celiac disease presented with generalized tonic-clonic seizures and hypocalcemia.
  • Following correction of her hypocalcemia, the patient's seizures ceased.
  • Electroencephalogram (EEG) revealed persistent occipital epileptiform activity despite seizure resolution.

Findings:

  • Hypocalcemia, particularly when secondary to malabsorption in celiac disease, significantly increases the risk of convulsions.

Related Experiment Videos

  • The case highlights a direct correlation between calcium levels and seizure activity in this patient population.
  • Persistent EEG abnormalities suggest potential long-term neurological impact even after metabolic correction.

    • Implications:

    • Early and thorough correction of hypocalcemia is vital in patients with celiac disease to mitigate seizure risk.
    • Physicians should consider screening for and managing hypocalcemia in celiac patients presenting with neurological symptoms.
    • This underscores the importance of addressing nutritional deficiencies in managing the extra-intestinal manifestations of celiac disease.