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Efficient secretion of biologically active mouse tumor necrosis factor alpha by Streptomyces lividans

L Van Mellaert1, C Dillen, P Proost

  • 1Laboratory of Microbiology, Rega Institute, K.U. Leuven, Belgium.

Gene
|December 2, 1994
PubMed

Insights

Streptomyces lividans efficiently secreted biologically active mouse tumor necrosis factor alpha (mTNF). This production method utilized a fusion protein strategy, overcoming processing challenges and yielding high-specific-activity mTNF.

Area of Science:

  • Biotechnology
  • Molecular Biology
  • Microbial Engineering

Background:

  • Recombinant protein production in microbial hosts is crucial for therapeutic applications.
  • Optimizing secretion and processing of foreign proteins in hosts like Streptomyces lividans presents challenges.
  • Mouse tumor necrosis factor alpha (mTNF) is a key cytokine with significant biological activity.

Purpose of the Study:

  • To engineer Streptomyces lividans for the high-level secretion of biologically active mouse tumor necrosis factor alpha (mTNF).
  • To investigate the processing and secretion mechanism of mTNF produced in S. lividans.
  • To characterize the specific activity and N-terminal heterogeneity of secreted mTNF.

Main Methods:

  • Fusion of mTNF cDNA to the alpha-amylase gene (aml) of Streptomyces venezuelae.
  • Expression and secretion of the fusion protein in Streptomyces lividans.
  • Quantification of secreted mTNF levels and specific activity assays.
  • N-terminal amino acid sequencing of secreted mTNF to analyze processing.

Main Results:

  • S. lividans secreted mTNF at moderately high levels (1-10 µg/ml).
  • The secreted mTNF exhibited high specific activity (1 x 10^8 units/mg protein) and was biologically active.
  • No unprocessed or significantly processed intracellular mTNF was detected.
  • N-terminal sequencing revealed microheterogeneity in secreted mTNF, attributed to aminopeptidase activity.

Conclusions:

  • Streptomyces lividans is a suitable host for the efficient secretion of biologically active mTNF.
  • The fusion strategy effectively directs secretion and ensures biological activity of mTNF.
  • Aminopeptidase activity in S. lividans contributes to the observed N-terminal microheterogeneity of secreted mTNF.

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