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Bilirubin and ascorbate antioxidant activity in neonatal plasma

V Gopinathan1, N J Miller, A D Milner

  • 1Free Radical Research Group, UMDS-Guy's Hospital, London, UK.

FEBS Letters
|August 1, 1994
PubMed

Insights

Premature infants have lower antioxidant capacity at birth. While ascorbate levels rise, total antioxidant status improves post-partum, with bilirubin playing a key antioxidant role.

Area of Science:

  • Neonatal Medicine
  • Biochemistry
  • Pediatric Research

Background:

  • Extremely low birth weight premature infants possess limited antioxidant defenses.
  • Antioxidant components not crossing the placenta until the third trimester impact neonatal status.

Purpose of the Study:

  • To compare total antioxidant activity and individual antioxidant concentrations in premature neonates versus term infants.
  • To investigate changes in antioxidant status and bilirubin levels post-partum.

Main Methods:

  • Plasma samples from premature neonates (27±2 weeks gestation) and term babies (38-41 weeks gestation) were analyzed.
  • Total antioxidant activity and specific antioxidant concentrations were measured.
  • Bilirubin levels were assessed in both groups.

Main Results:

  • Premature neonates exhibited elevated plasma ascorbate at birth but lower overall antioxidant status compared to term infants.
  • Post-partum, ascorbate levels normalized, and plasma bilirubin increased significantly in both groups.
  • Premature neonates showed increased total plasma antioxidant status by 5 days post-partum.

Conclusions:

  • Bilirubin is a significant plasma antioxidant, as evidenced by its direct correlation with total antioxidant activity in term infants.
  • Premature infants' antioxidant capacity undergoes dynamic changes post-birth, with bilirubin contributing to overall status.

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