Related Experiment Videos

Expression and localization of tissue factor-based procoagulant activity (PCA) in pigeon monocyte-derived macrophages

M Gupta1, G J Doellgast, T Cheng

  • 1Department of Pathology, Bowman Gray School of Medicine, Wake Forest University, Winston-Salem, North Carolina 27157-1092.

Thrombosis and Haemostasis
|December 20, 1993
PubMed

Insights

Macrophages in White Carneau pigeons exhibit tissue factor-based procoagulant activity after endotoxin stimulation. This finding suggests a role for these cells in initiating fibrin polymerization and thrombotic complications of atherosclerosis.

Area of Science:

  • Cardiovascular Biology
  • Immunology
  • Hematology

Background:

  • Monocyte-derived macrophages are central to atherosclerosis initiation and progression.
  • These cells have been linked to thrombotic complications in cardiovascular disease.

Purpose of the Study:

  • To investigate tissue factor-based procoagulant activity in cultured macrophages from White Carneau pigeons.
  • To explore the role of this activity in the pathobiology of atherosclerosis and its thrombotic complications.

Main Methods:

  • Cultured macrophages from White Carneau pigeons were stimulated with endotoxin.
  • Enzyme-Linked Coagulation Assay (ELCA) was used to measure procoagulant activity.
  • Immunogold labeling with a polyclonal antibody specific to human tissue factor detected tissue factor antigen.

Main Results:

  • Endotoxin stimulation induced significant tissue factor-based procoagulant activity in pigeon macrophages.
  • This activity was comparable to that observed in pigeon brain homogenate.
  • Tissue factor antigen was uniformly distributed on the plasma membrane of stimulated macrophages.
  • Procoagulant activity was detectable with as few as 1-3 cells.

Conclusions:

  • Cultured pigeon macrophages possess potent procoagulant activity mediated by tissue factor.
  • This macrophage-derived procoagulant activity may contribute to fibrin polymerization and thrombotic events in atherosclerosis.
  • These findings highlight a potential mechanism in the pathobiology of atherosclerosis in this avian model.

Related Concept Videos