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Cyclosporin-A-induced gingival overgrowth in renal transplant children

B Wondimu1, G Dahllöf, U Berg

  • 1Department of Pedodontics, Faculty of Odontology, Huddinge University Hospital, Karolinska Institutet, Sweden.

Scandinavian Journal of Dental Research
|October 1, 1993
PubMed

Insights

Cyclosporin-A (CsA) can cause gingival overgrowth in children after kidney transplants. Higher initial CsA doses increase this risk, highlighting the importance of monitoring drug administration.

Area of Science:

  • Pediatric Nephrology
  • Oral Medicine
  • Pharmacology

Background:

  • Renal transplant recipients often require immunosuppression.
  • Cyclosporin-A (CsA) is a common immunosuppressant used in pediatric transplantation.
  • Gingival overgrowth is a known side effect of CsA therapy.

Purpose of the Study:

  • To assess the prevalence and characteristics of gingival overgrowth in pediatric renal transplant patients on CsA.
  • To investigate the relationship between CsA dosage and the development of gingival overgrowth.

Main Methods:

  • A cohort of pediatric renal transplant recipients on CsA for at least 12 months was studied.
  • Data collected included demographics, transplant duration, CsA dosage (initial and current), and oral health parameters (plaque, inflammation, attachment loss, gingival overgrowth).
  • Gingival overgrowth was defined by increased sulcus probing depth (pseudopockets).

Main Results:

  • Thirteen percent of the children exhibited gingival overgrowth.
  • Children with gingival overgrowth had received a significantly higher total CsA dose during the first six posttransplant months compared to those without overgrowth.
  • No significant difference was noted in current oral CsA dose or blood trough levels between groups.

Conclusions:

  • The development of CsA-induced gingival overgrowth in pediatric renal transplant patients is positively associated with the cumulative CsA dose administered in the early posttransplant period.
  • Early high-dose CsA administration may be a critical factor in the pathogenesis of gingival overgrowth in this population.

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