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Secondary osteoporosis

I T Boyle1

  • 1University Department of Medicine, Glasgow Royal Infirmary, UK.

Bailliere'S Clinical Rheumatology
|October 1, 1993
PubMed
Summary
This summary is machine-generated.

Secondary osteoporosis, a common complication of chronic diseases, increases fracture risk. Management involves treating underlying conditions, optimizing lifestyle, and considering bone-active drugs for prevention and bone conservation.

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Area of Science:

  • Endocrinology
  • Bone Metabolism
  • Geriatrics

Background:

  • Osteoporosis, characterized by increased fracture risk, frequently complicates chronic diseases.
  • Chronic illnesses impact lifestyle, reducing physical activity and gravitational bone strain.
  • Gastrointestinal issues and altered sex hormone status (hypogonadism) are critical factors in secondary osteoporosis.

Purpose of the Study:

  • To highlight the prevalence and management of secondary osteoporosis in patients with chronic diseases.
  • To emphasize the role of hormone status and lifestyle factors in bone health.
  • To discuss therapeutic strategies, including hormone replacement and anti-resorptive agents.

Main Methods:

  • Review of literature on secondary osteoporosis associated with chronic diseases.

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  • Analysis of the impact of lifestyle, nutrition, and hormonal factors on bone density.
  • Evaluation of current and emerging pharmacological treatments for osteoporosis.

    • Main Results:

    • Secondary osteoporosis is prevalent across various chronic conditions, affecting bone nutrition and structure.
    • Hormone replacement therapy (estrogen, testosterone) is beneficial for bone conservation in hypo-oestrogenic women and hypogonadal men.
    • Bisphosphonates and other anti-bone-resorbing drugs offer effective treatment for steroid-induced osteoporosis, with prophylaxis becoming a possibility.

    Conclusions:

    • A comprehensive approach to secondary osteoporosis includes identifying underlying causes, optimizing lifestyle, and considering hormone therapy and bone-active drugs.
    • Early detection via bone densitometry is crucial, especially with increased patient lifespan.
    • Spontaneous bone restoration is possible in some cases (e.g., thyrotoxicosis, Cushing's syndrome) after resolving the primary condition.