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Detection of measles virus nucleoprotein mRNA in autopsied brain tissues

Y Katayama1, H Hotta, A Nishimura

  • 1Department of Microbiology, Kobe University School of Medicine, Hyogo, Japan.

Insights

Measles virus (MV) RNA was found in the brains of 18% of individuals without neurological symptoms. Detected MV sequences had mutations, suggesting persistent, low-replication infections in the human brain.

Area of Science:

  • Virology
  • Neuroscience
  • Molecular Biology

Background:

  • Measles virus (MV) is a highly contagious pathogen that can cause acute illness.
  • Subacute sclerosing panencephalitis (SSPE) is a rare, fatal neurological complication of measles infection.
  • The persistence of measles virus in the central nervous system without overt disease is not well understood.

Purpose of the Study:

  • To investigate the prevalence of measles virus (MV) persistent infection in the human brain.
  • To analyze the genetic characteristics of detected MV sequences in brain tissue.
  • To explore the potential mechanisms behind asymptomatic MV persistence in the central nervous system.

Main Methods:

  • Real-time polymerase chain reaction (RT-PCR) was used to detect measles virus (MV) nucleoprotein (NP) mRNA in brain tissue samples.
  • Nucleotide sequences of detected MV NP genes were analyzed for genetic variations.
  • Comparison of brain-derived MV sequences with wild-type MV and SSPE virus isolates.

Main Results:

  • Measles virus (MV) mRNA was detected in 11 out of 61 (18%) brain tissue samples from individuals without SSPE-like symptoms.
  • The majority of detected MV nucleoprotein (NP) sequences exhibited significant asynonymous nucleotide substitutions compared to wild-type MV and SSPE virus.
  • These findings indicate the presence of genetically distinct MV strains persisting in the brain.

Conclusions:

  • Measles virus (MV) can persist asymptomatically in the human brain.
  • The detected MV strains in the brain show genetic divergence from typical wild-type and SSPE strains.
  • Reduced viral replication may be a key factor in the asymptomatic persistence of MV in the central nervous system.

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