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Mitochondria changes in human muscle after prolonged exercise, endurance training and selenium supplementation
A J Zamora1, F Tessier, P Marconnet
1CNRS, Faculté des Sciences du Sport, Université de la Méditerranée, Marseille, France.
Summary
Selenium supplementation may dampen exercise-induced changes in muscle mitochondria. Endurance training increased mitochondria quantity, while acute exercise caused swelling, effects potentially mitigated by selenium.
Area of Science:
- Exercise Physiology
- Mitochondrial Biology
- Nutritional Biochemistry
Background:
- Muscle mitochondria are crucial for energy production.
- Exercise training and acute exercise impact mitochondrial structure and function.
- Selenium's role in exercise adaptation is not fully understood.
Purpose of the Study:
- To investigate the effects of endurance training and acute exercise on muscle mitochondria.
- To determine the influence of selenium supplementation on these responses.
- To analyze mitochondrial structural changes in response to exercise and selenium.
Main Methods:
- Double-blind study with 24 young males over 10 weeks.
- Endurance training program with or without selenium supplementation.
- Muscle biopsies analyzed via electron microscopy for mitochondrial quantification (QA, AA, â).
Main Results:
- Training significantly increased mitochondrial number (QA) and area (AA).
- Selenium supplementation with training led to increased mean mitochondrial area (â).
- Acute exercise caused mitochondrial swelling (increased â), an effect modulated by training and selenium.
Conclusions:
- Endurance training enhances mitochondrial quantity and size.
- Selenium may attenuate exercise-induced mitochondrial alterations.
- Muscle mitochondria exhibit significant short-term plasticity in response to acute exercise.