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Transcription factors of the NFAT family: regulation and function

A Rao1, C Luo, P G Hogan

  • 1Center for Blood Research, Harvard Medical School, Boston, Massachusetts 02115, USA. arao@cbr.med.harvard.edu

Insights

Nuclear factor of activated T cells (NFAT) proteins are key regulators of immune responses, controlled by calcineurin. This review covers NFAT diversity, regulation, and cooperation with other factors like AP-1 for gene expression.

Area of Science:

  • Immunology
  • Molecular Biology
  • Cell Biology

Background:

  • Nuclear factor of activated T cells (NFAT) proteins are crucial transcription factors in immune cells.
  • NFATs regulate genes essential for immune responses, including cytokine production.
  • Their activity is modulated by calcineurin, a target of immunosuppressive drugs like cyclosporin A and FK506.

Purpose of the Study:

  • To review recent advancements in understanding the NFAT family of transcription factors.
  • To elucidate the regulatory mechanisms governing NFAT protein activity within cells.
  • To explore the cooperative interactions of NFAT proteins with other transcription factors.

Main Methods:

  • Literature review of recent data on NFAT.
  • Analysis of NFAT protein regulation by calcineurin.
  • Examination of NFAT interactions with Rel and AP-1 transcription factors.

Main Results:

  • NFAT proteins are diverse and critical for immune cell gene transcription.
  • Calcineurin tightly regulates NFAT translocation to the nucleus.
  • NFAT proteins cooperate with AP-1 (Fos/Jun) and Rel-family proteins at composite regulatory sites.

Conclusions:

  • NFAT transcription factors are central to immune system regulation.
  • Understanding NFAT regulation and cooperation is vital for immune response research.
  • NFATs, in conjunction with other factors, control inducible gene expression in immune cells.

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