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Rabies

T Hemachudha1, P Phuapradit

  • 1Department of Medicine, Chulalongkorn University Hospital, Bangkok, Thailand. fmedthm@chulkn.car.chula.ac.th

Current Opinion in Neurology
|June 1, 1997
PubMed
Summary
This summary is machine-generated.

Rabies mechanisms remain unclear, with varied clinical signs in furious and dumb types. Bat rabies variants and survivor presentations highlight diverse symptoms, possibly due to cellular tropism and neurotransmitter differences.

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Area of Science:

  • Neurology
  • Virology
  • Infectious Diseases

Background:

  • Rabies presents complex clinical manifestations, including furious and dumb types, with poorly understood fatal mechanisms.
  • Clinical symptomatology of rabies can be variable, especially in cases linked to insectivorous or frugivorous bat virus exposure.

Purpose of the Study:

  • To review classic and nonclassic clinical features of canine and bat rabies variants.
  • To discuss atypical presentations observed in rabies survivors.

Main Methods:

  • Literature review of classic and contemporary studies on rabies clinical presentations.
  • Analysis of case reports and epidemiological data concerning bat-associated rabies.

Main Results:

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  • Summarizes diverse clinical features across different rabies variants (canine, bat).
  • Highlights atypical presentations in rabies survivors, challenging traditional symptomatology.
  • Identifies potential factors like cellular tropism, spread routes, and neurotransmitter modulation for symptom variability.

    • Conclusions:

    • Variability in rabies clinical presentation may stem from differences in cellular tropism and spread pathways.
    • Neurotransmitter alterations likely contribute to diverse neurobehavioral and neuroendocrine-immune responses in rabies.