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Effect of octreotide, captopril or insulin on renal changes and UAE in long-term experimental diabetes

H Grønbaek1, I Vogel, R Osterby

  • 1Institute of Experimental Clinical Research, Aarhus University Hospital, Denmark.

Kidney International
|February 7, 1998
PubMed

Insights

Diabetic kidney disease shows increased renal growth and albuminuria. Insulin normalized these changes, while octreotide and captopril reduced renal size and albuminuria without affecting blood glucose.

Area of Science:

  • Nephrology
  • Endocrinology
  • Pharmacology

Background:

  • Diabetes mellitus is associated with progressive renal and glomerular hypertrophy.
  • Diabetic nephropathy often presents with increased urinary albumin excretion (UAE).
  • Angiotensin-converting enzyme (ACE) inhibitors and somatostatin analogs show potential in managing experimental diabetic nephropathy.

Purpose of the Study:

  • To evaluate the effects of octreotide and captopril, alone or in combination, on established diabetic renal changes.
  • To compare these effects with insulin treatment in a model of experimental diabetes.

Main Methods:

  • Induction of diabetes in experimental models.
  • Administration of octreotide, captopril, their combination, or insulin for three weeks after three months of untreated diabetes.
  • Assessment of renal and glomerular size, urinary albumin excretion (UAE), and blood glucose levels.

Main Results:

  • Diabetes significantly increased renal and glomerular size and UAE.
  • Octreotide and captopril reduced renal size but not glomerular size; combined therapy reduced UAE.
  • Insulin treatment normalized renal and glomerular size and UAE by achieving euglycemia.
  • Neither octreotide nor captopril affected blood glucose levels.

Conclusions:

  • Insulin therapy, by normalizing metabolic derangements, effectively reverses established diabetic renal and glomerular hypertrophy and reduces UAE.
  • Combined octreotide and captopril treatment demonstrates efficacy in reducing renal growth and UAE in diabetic animals, independent of glycemic control.

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