Jove
Visualize
Contact Us
JoVE
x logofacebook logolinkedin logoyoutube logo
ABOUT JoVE
OverviewLeadershipBlogJoVE Help Center
AUTHORS
Publishing ProcessEditorial BoardScope & PoliciesPeer ReviewFAQSubmit
LIBRARIANS
TestimonialsSubscriptionsAccessResourcesLibrary Advisory BoardFAQ
RESEARCH
JoVE JournalMethods CollectionsJoVE Encyclopedia of ExperimentsArchive
EDUCATION
JoVE CoreJoVE BusinessJoVE Science EducationJoVE Lab ManualFaculty Resource CenterFaculty Site
Terms & Conditions of Use
Privacy Policy
Policies

Related Experiment Videos

Behavior genetics and eating disorders

J K Hewitt1

  • 1Institute for Behavioral Genetics, University of Colorado, Boulder 80303, USA.

Psychopharmacology Bulletin
|January 1, 1997
PubMed
Summary
This summary is machine-generated.

Related Concept Videos

You might also read

Related Articles

Articles linked to this work by shared authors, journal, and citation graph.

Sort by
Same author

Substance use patterns in 9-10 year olds: Baseline findings from the adolescent brain cognitive development (ABCD) study.

Drug and alcohol dependence·2021
Same author

Baseline brain function in the preadolescents of the ABCD Study.

Nature neuroscience·2021
Same author

Genome-wide association study of lifetime cannabis use based on a large meta-analytic sample of 32 330 subjects from the International Cannabis Consortium.

Translational psychiatry·2016
Same author

Stability and change of genetic and environmental effects on the common liability to alcohol, tobacco, and cannabis DSM-IV dependence symptoms.

Behavior genetics·2013
Same author

Nominal association with CHRNA4 variants and nicotine dependence.

Genes, brain, and behavior·2013
Same author

The heritability of general cognitive ability increases linearly from childhood to young adulthood.

Molecular psychiatry·2009
Same journal

Catatonia Associated with Psychosis: A Retrospective Study of 164 Inpatients.

Psychopharmacology bulletin·2026
Same journal

Escitalopram-Induced Spasmodic Torticollis in an Early Psychosis Service: A Case Report.

Psychopharmacology bulletin·2026
Same journal

Beyond Exposure: Standardizing Buprenorphine Training in Pain Medicine Fellowship.

Psychopharmacology bulletin·2026
Same journal

Association of CBC-Derived Indices of Inflammation with Response to Glecaprevir/Pibrentasvir for PTSD.

Psychopharmacology bulletin·2026
Same journal

When Care Becomes Dangerous: Patient Assaults Against Physicians.

Psychopharmacology bulletin·2026
Same journal

Efficacy and Safety of Centanafadine in Attention-Deficit/Hyperactivity Disorder: A Systematic Review and Meta-Analysis of Randomized Controlled Trials.

Psychopharmacology bulletin·2026
See all related articles

Behavior genetics research shows that while genes influence eating disorder vulnerability, environmental factors are crucial. Understanding these complex interactions is key to identifying specific genetic and environmental risk factors.

Area of Science:

  • Behavioral genetics
  • Psychiatry
  • Genetics

Background:

  • Eating disorders develop through complex interactions between individual behaviors and environmental circumstances.
  • Simple genetic explanations for eating disorders are insufficient due to multifactorial influences.
  • Behavior genetics investigates genetic and environmental contributions to individual differences in eating disorder vulnerability.

Purpose of the Study:

  • To review the role of behavior genetics in understanding eating disorder vulnerability.
  • To highlight the importance of considering gene-environment interactions in eating disorder research.
  • To guide future research in identifying specific genetic and environmental risk factors.

Main Methods:

  • Review of twin, family, and adoption studies.

Related Experiment Videos

  • Analysis of heritability and environmental responsiveness of eating disorder phenotypes.
  • Discussion of approaches for identifying genetic and environmental risk factors.

    • Main Results:

    • Consistent evidence supports genetic factors influencing eating disorder vulnerability.
    • The precise genetic mechanisms and their interaction with environmental factors remain unclear.
    • Twin, family, and adoption studies help distinguish heritable traits from environmentally influenced ones.

    Conclusions:

    • Genetic factors play a role in eating disorder vulnerability, but environmental influences are significant.
    • Further research is needed to elucidate the complex interplay of genes and environment.
    • Developing dimensional indices of vulnerability will enhance genetic research on eating disorders.