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Prothrombin fragment 1+2 is a risk factor for myocardial infarction in treated hypertensive men

S Agewall1, J Wikstrand, B Fagerberg

  • 1Department of Medicine, Sahlgrenska University Hospital, Göteborg University, Sweden.

Journal of Hypertension
|October 31, 1998
PubMed

Insights

Prothrombin fragment 1+2 and C-reactive protein levels predict major coronary events in hypertensive men. These hemostatic factors also predict mortality, highlighting their importance in cardiovascular disease risk.

Area of Science:

  • Cardiovascular Medicine
  • Hematology
  • Biomarkers

Background:

  • Hemostatic factors are implicated in acute coronary heart disease development.
  • Understanding these factors is crucial for predicting cardiovascular events.

Purpose of the Study:

  • To assess fibrinogen, von Willebrand factor, prothrombin fragment 1+2, thrombin-antithrombin complex, plasminogen activator inhibitor activity, and C-reactive protein as predictors of major coronary events.
  • To evaluate these markers' association with mortality in a cohort of hypertensive men.

Main Methods:

  • A cohort of 131 men (aged 56-77) with treated hypertension and additional cardiovascular risk factors were studied.
  • Mean follow-up duration was 3.0 years, with monitoring for major coronary events (myocardial infarction, sudden death) and mortality.
  • Statistical adjustments were made for other known cardiovascular risk factors.

Main Results:

  • Prothrombin fragment 1+2 and C-reactive protein emerged as independent predictors of major coronary events.
  • Fibrinogen and prothrombin fragment 1+2 levels were identified as independent predictors of mortality.
  • Other measured hemostatic variables did not show significant associations with major coronary events.

Conclusions:

  • Prothrombin fragment 1+2 and C-reactive protein levels are significant independent predictors of major coronary events in treated hypertensive men.
  • These biomarkers may aid in risk stratification for cardiovascular events and mortality in this population.
Abstract

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