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Published on: August 18, 2016
What makes a coronary myocardial bridge symptomatic?
F J Podbielski1, R el Chaer, M G Massad
1Division of Cardiothoracic Surgery, University of Illinois at Chicago, USA.
Insights
Coronary myocardial bridging can cause exertional chest pain. This case study shows non-invasive treatment with fluids and calcium channel blockers effectively resolved symptoms and improved coronary artery flow.
Area of Science:
- Cardiology
- Cardiovascular Medicine
- Interventional Cardiology
Background:
- Coronary myocardial bridging is a congenital anomaly where a segment of a coronary artery travels through the heart muscle.
- Its clinical significance and optimal management strategies remain subjects of ongoing debate.
- Various interventional approaches have been proposed for symptomatic patients.
Observation:
- A patient presented with exertional chest pain, diaphoresis, and ischemic electrocardiogram changes.
- Cardiac catheterization revealed significant systolic narrowing in the mid left anterior descending coronary artery, indicative of myocardial bridging.
- The patient experienced acute anterior myocardial wall ischemia.
Findings:
- Non-invasive management including intravenous fluid resuscitation and calcium channel blockade was initiated.
- These interventions led to the normalization of the patient's electrocardiogram.
- Complete resolution of anginal symptoms and improvement in coronary angiographic findings were observed.
Implications:
- This case highlights the potential efficacy of conservative medical management for symptomatic coronary myocardial bridging.
- It suggests that interventions like fluid resuscitation and calcium channel blockers can alleviate ischemia and improve coronary flow.
- Further research into non-invasive treatment protocols for myocardial bridging is warranted.
Abstract:
The clinical significance of coronary myocardial bridging has been debated and different interventional strategies proposed. We discuss the case of a patient who presented with the recent onset of symptoms of exertional chest pain, diaphoresis, and electrocardiographic signs of acute anterior myocardial wall ischemia. Cardiac catheterization demonstrated a segmental high-grade narrowing of the mid left anterior descending coronary artery during systole consistent with bridging. Intravenous fluid resuscitation and calcium channel blockade resulted in normalization of his electrocardiogram and complete resolution of his anginal symptoms and coronary angiographic systolic narrowing.
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