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Reflex sympathetic dystrophy precipitated by brachial plexitis
1Department of Neurology, EMG Laboratory, Hospital of Santa Maria, Lisbon, Portugal. mamedemg@mail.telepac.pt
Electromyography and Clinical Neurophysiology
|December 8, 1998
Summary
Reflex sympathetic dystrophy, a condition of sympathetic nervous system overactivity, can arise after brachial plexitis. Abnormal nerve sodium channels may trigger this complication during reinnervation.
Area of Science:
- Neurology
- Neuroscience
- Autonomic Nervous System Research
Background:
- Reflex sympathetic dystrophy (RSD), now known as complex regional pain syndrome (CRPS), is characterized by sympathetic nervous system hyperactivity.
- While commonly linked to local trauma, its association with brachial plexitis (neuralgic amyotrophy) is rarely documented.
Observation:
- This report details a novel case of reflex sympathetic dystrophy following brachial plexitis.
- The patient's presentation highlights a potential, albeit infrequent, complication of this neurological condition.
Findings:
- Abnormal sodium channels in reinnervating somatic nerve fibers may develop ectopic pacemaker activity.
- Sympathetic efferent impulses can stimulate these abnormal channels, triggering RSD during the nerve reinnervation phase.
- Severe brachial plexitis increases the risk of developing this complication due to nerve fiber changes.
Implications:
- Neurologists must recognize the potential link between brachial plexitis and reflex sympathetic dystrophy.
- Awareness of this association is crucial for timely diagnosis and appropriate patient management.
- Further research into the neurophysiological mechanisms underlying this association is warranted.