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Current Medicinal Chemistry. Cardiovascular and Hematological Agents|August 24, 2004
Bone marrow reconstitution as a relevant model of genetically programmed leukemiaA Dolnikov, S Shen, T Passioura, et al.Current Issues in Molecular Biology|May 4, 2004
Cancer gene suppression strategies: issues and potentialT Passioura, G SymondsCancer Gene Therapy|December 29, 2000
Induced p21WAF1 expression acts to reverse myc myelomonocytic cell transformationA Dolnikov, M Millington, L Q Sun, et al.Oncogene|March 21, 1996
Apoptosis in v-myc transformation of myelomonocytic cells and its modulation by CSF-1A Dolnikov, R L Ward, N J Hawkins, et al.Leukemia|April 29, 1998
V-myc in a simple, single gene retroviral vector causes rapid induction of leukemia and concomitant apoptosis following bone marrow transplantationA Dolnikov, Y Shounan, M Millington, et al.Journal of Hematotherapy & Stem Cell Research|November 25, 2000
The beta-galactosidase gene as a marker for hematopoietic reconstitution: individual cell analysis in a murine bone marrow transplantation modelA Dolnikov, M Millington, E Beruter, et al.International Journal of Oncology|May 11, 2011
Transformation by v-erb-B - induction of apoptosis is abrogated in a myeloid cell-lineA Dolnikov, Y Shounan, N Hawkins, et al.Leukemia|October 27, 1997
Myeloproliferative disease and myelodysplastic syndrome induced by transplantation of bone marrow cells expressing mutant p53Y Shounan, K MacKenzie, A Dolnikov, et al.Blood|March 9, 1999
Mutant N-ras induces myeloproliferative disorders and apoptosis in bone marrow repopulated miceK L MacKenzie, A Dolnikov, M Millington, et al.Cancer Gene Therapy|September 1, 1996
Ribozyme-mediated suppression of v-myc expression abrogates apoptosis in transformed monocytesA Dolnikov, A King, C Luxford, et al.Pageof 91