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Abdul M Mondal

Showing results (1-10 of 9) with videos related to

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Journal of Medical Virology|June 2, 2020
Conditional cell reprogramming for modeling host-virus interactions and human viral diseasesXuefeng Liu, Abdul M Mondal
Journal of Biochemical and Molecular Toxicology|November 24, 2022
Crosstalk of moderate ROS and PARP-1 contributes to sustainable proliferation of conditionally reprogrammed keratinocytesLinhua Liu, Abdul M Mondal, Xuefeng Liu
Nature Communications|August 22, 2014
Autophagic degradation of the inhibitory p53 isoform Δ133p53α as a regulatory mechanism for p53-mediated senescenceIzumi Horikawa, Kaori Fujita, Lisa M Miller Jenkins, et al.
Nature Cell Biology|November 9, 2010
Positive feedback between p53 and TRF2 during telomere-damage signalling and cellular senescenceKaori Fujita, Izumi Horikawa, Abdul M Mondal, et al.
Cell Death & Disease|July 5, 2018
Δ133p53α, a natural p53 isoform, contributes to conditional reprogramming and long-term proliferation of primary epithelial cellsAbdul M Mondal, Hua Zhou, Izumi Horikawa, et al.
Cell Death and Differentiation|April 1, 2017
Δ133p53 represses p53-inducible senescence genes and enhances the generation of human induced pluripotent stem cellsIzumi Horikawa, Kye-Yoon Park, Kazunobu Isogaya, et al.
Biochemical and Biophysical Research Communications|July 16, 2019
Fidelity of a PDX-CR model for bladder cancerAbdul M Mondal, Ai-Hong Ma, Guangzhao Li, et al.
The Journal of Clinical Investigation|November 16, 2013
p53 isoforms regulate aging- and tumor-associated replicative senescence in T lymphocytesAbdul M Mondal, Izumi Horikawa, Sharon R Pine, et al.
Nature Cell Biology|August 25, 2009
p53 isoforms Delta133p53 and p53beta are endogenous regulators of replicative cellular senescenceKaori Fujita, Abdul M Mondal, Izumi Horikawa, et al.
Pageof 1

Showing results (1-10 of 9) with videos related to

Sort By:
Pageof 1
Journal of Medical Virology|June 2, 2020
Conditional cell reprogramming for modeling host-virus interactions and human viral diseasesXuefeng Liu, Abdul M Mondal
Journal of Biochemical and Molecular Toxicology|November 24, 2022
Crosstalk of moderate ROS and PARP-1 contributes to sustainable proliferation of conditionally reprogrammed keratinocytesLinhua Liu, Abdul M Mondal, Xuefeng Liu
Nature Communications|August 22, 2014
Autophagic degradation of the inhibitory p53 isoform Δ133p53α as a regulatory mechanism for p53-mediated senescenceIzumi Horikawa, Kaori Fujita, Lisa M Miller Jenkins, et al.
Nature Cell Biology|November 9, 2010
Positive feedback between p53 and TRF2 during telomere-damage signalling and cellular senescenceKaori Fujita, Izumi Horikawa, Abdul M Mondal, et al.
Cell Death & Disease|July 5, 2018
Δ133p53α, a natural p53 isoform, contributes to conditional reprogramming and long-term proliferation of primary epithelial cellsAbdul M Mondal, Hua Zhou, Izumi Horikawa, et al.
Cell Death and Differentiation|April 1, 2017
Δ133p53 represses p53-inducible senescence genes and enhances the generation of human induced pluripotent stem cellsIzumi Horikawa, Kye-Yoon Park, Kazunobu Isogaya, et al.
Biochemical and Biophysical Research Communications|July 16, 2019
Fidelity of a PDX-CR model for bladder cancerAbdul M Mondal, Ai-Hong Ma, Guangzhao Li, et al.
The Journal of Clinical Investigation|November 16, 2013
p53 isoforms regulate aging- and tumor-associated replicative senescence in T lymphocytesAbdul M Mondal, Izumi Horikawa, Sharon R Pine, et al.
Nature Cell Biology|August 25, 2009
p53 isoforms Delta133p53 and p53beta are endogenous regulators of replicative cellular senescenceKaori Fujita, Abdul M Mondal, Izumi Horikawa, et al.
Pageof 1