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Toxicologic Pathology|March 17, 2007
Gene expression analysis offers unique advantages to histopathology in liver biopsy evaluationsAlexandra N Heinloth, Gary A Boorman, Julie F Foley, et al.
Pain Research and Treatment|September 14, 2012
Review of Efficacy and Safety of Duloxetine 40 to 60 mg Once Daily in Patients with Diabetic Peripheral Neuropathic PainVladimir Skljarevski, Elijah P Frakes, Doron Sagman, et al.
Psychiatry Research|June 26, 2012
Evaluation of genetic models for response in a randomized clinical trial of duloxetine in major depressive disorderJohn P Houston, Wei Zou, Virginie Aris, et al.
Toxicologic Pathology|December 13, 2006
Optimal sampling of rat liver tissue for toxicogenomic studiesJulie F Foley, Jennifer B Collins, David M Umbach, et al.
BMC Psychiatry|March 31, 2009
Predictors and correlates for weight changes in patients co-treated with olanzapine and weight mitigating agents; a post-hoc analysisVirginia L Stauffer, Ilya Lipkovich, Vicki Poole Hoffmann, et al.
Toxicologic Pathology|June 24, 2004
Application of toxicogenomics to toxicology: basic concepts in the analysis of microarray dataRichard D Irwin, Gary A Boorman, Michael L Cunningham, et al.
The Journal of Clinical Psychiatry|April 7, 2012
Association of common variations in the norepinephrine transporter gene with response to olanzapine-fluoxetine combination versus continued-fluoxetine treatment in patients with treatment-resistant depression: a candidate gene analysisJohn P Houston, Kit Lau, Virginie Aris, et al.
Postgraduate Medicine|July 26, 2008
Answers to the most common questions about the hepatic safety profile of duloxetineMadelaine M Wohlreich, Nayan Acharya, Indiana Strombom, et al.
Therapeutics and Clinical Risk Management|February 12, 2009
Double-blind, randomized trial comparing efficacy and safety of continuing olanzapine versus switching to quetiapine in overweight or obese patients with schizophrenia or schizoaffective disorderWalter Deberdt, Ilya Lipkovich, Alexandra N Heinloth, et al.
Molecular and Cellular Biology|November 26, 2002
An ATR- and Chk1-dependent S checkpoint inhibits replicon initiation following UVC-induced DNA damageTimothy P Heffernan, Dennis A Simpson, Alexandra R Frank, et al.
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