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International Journal of Molecular Sciences|March 27, 2020
VLA-4 Expression and Activation in B Cell Malignancies: Functional and Clinical AspectsAndrea Härzschel, Antonella Zucchetto, Valter Gattei, et al.International Journal of Molecular Sciences|December 19, 2018
Rac GTPases in Hematological MalignanciesValerie Durand-Onaylı, Theresa Haslauer, Andrea Härzschel, et al.Cancers|April 11, 2020
An Updated Perspective on Current Prognostic and Predictive Biomarkers in Chronic Lymphocytic Leukemia in the Context of Chemoimmunotherapy and Novel Targeted TherapyJared A Cohen, Riccardo Bomben, Federico Pozzo, et al.JTO Clinical and Research Reports|April 13, 2026
Treatment Patterns, Outcome, and Quality of Life of Patients With Extensive-Stage SCLC Receiving Third-Line Therapy-Data From the German CRISP Registry (AIO-TRK-0315): A Brief ReportMaximilian Rost, Rieke Fischer, Martin Reck, et al.Journal of Leukocyte Biology|December 10, 2021
Kindlin-3 maintains marginal zone B cells but confines follicular B cell activation and differentiationAndrea Härzschel, Lixia Li, Peter W Krenn, et al.Haematologica|July 4, 2020
CD44 engagement enhances acute myeloid leukemia cell adhesion to the bone marrow microenvironment by increasing VLA-4 avidityJulia C Gutjahr, Elisabeth Bayer, Xiaobing Yu, et al.The Journal of Experimental Medicine|January 6, 2018
Functional and clinical relevance of VLA-4 (CD49d/CD29) in ibrutinib-treated chronic lymphocytic leukemiaErika Tissino, Dania Benedetti, Sarah E M Herman, et al.Leukemia|August 14, 2024
The VLA-4 integrin is constitutively active in circulating chronic lymphocytic leukemia cells via BCR autonomous signaling: a novel anchor-independent mechanism exploiting soluble blood-borne ligandsErika Tissino, Annalisa Gaglio, Antonella Nicolò, et al.Blood|March 6, 2026
CD49d governs immune synapse formation through actin rearrangements and synchronizes BCR signaling in CLLLaura Polcik, Abhishek Pethe, Driti Ashok, et al.Pageof 1