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Molecular Cell|August 4, 2006
Head module control of mediator interactionsYuichiro Takagi, Guillermo Calero, Hirofumi Komori, et al.Pain|January 11, 2018
Disruption of nNOS-NOS1AP protein-protein interactions suppresses neuropathic pain in miceWan-Hung Lee, Li-Li Li, Aarti Chawla, et al.The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|March 21, 2008
Phosphorylation of sodium channel Na(v)1.8 by p38 mitogen-activated protein kinase increases current density in dorsal root ganglion neuronsAndy Hudmon, Jin-Sung Choi, Lynda Tyrrell, et al.Molecular and Cellular Neurosciences|July 28, 2014
Regulation of voltage-gated Ca(2+) currents by Ca(2+)/calmodulin-dependent protein kinase II in resting sensory neuronsSandra Kostic, Bin Pan, Yuan Guo, et al.The Journal of Physiology|July 25, 2012
The human Nav1.5 F1486 deletion associated with long QT syndrome leads to impaired sodium channel inactivation and reduced lidocaine sensitivityWeihua Song, Yucheng Xiao, Hanying Chen, et al.Biochemistry|July 3, 2007
Displacement of alpha-actinin from the NMDA receptor NR1 C0 domain By Ca2+/calmodulin promotes CaMKII bindingMichelle A Merrill, Zulfiqar Malik, Zeynep Akyol, et al.Scientific Reports|April 16, 2016
Novel Roles for Peroxynitrite in Angiotensin II and CaMKII SignalingChaoming Zhou, Swarna S Ramaswamy, Derrick E Johnson, et al.Neuropharmacology|June 14, 2015
Small molecule inhibitors of PSD95-nNOS protein-protein interactions as novel analgesicsWan-Hung Lee, Zhili Xu, Nicole M Ashpole, et al.Proceedings of the National Academy of Sciences of the United States of America|August 29, 2019
Assemblies of calcium/calmodulin-dependent kinase II with actin and their dynamic regulation by calmodulin in dendritic spinesQian Wang, Mingchen Chen, Nicholas P Schafer, et al.The Journal of Biological Chemistry|April 20, 2012
Ca2+/calmodulin-dependent protein kinase II (CaMKII) regulates cardiac sodium channel NaV1.5 gating by multiple phosphorylation sitesNicole M Ashpole, Anthony W Herren, Kenneth S Ginsburg, et al.Pageof 5