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Hematology. American Society of Hematology. Education Program|December 10, 2017
Evolving treatment paradigms in sickle cell diseaseRamasamy Jagadeeswaran, Angela Rivers
Seminars in Perinatology|January 27, 2009
Congenital cytopenias and bone marrow failure syndromesAngela Rivers, William B Slayton
American Journal of Physiology. Regulatory, Integrative and Comparative Physiology|August 2, 2018
Potential role of LSD1 inhibitors in the treatment of sickle cell disease: a review of preclinical animal model dataAngela Rivers, Ramasamy Jagadeeswaran, Donald Lavelle
Seminars in Hematology|June 3, 2026
Evolving insights into mitochondrial presence in RBC in sickle cell disease: Red blood cell and reticulocyte pathophysiology, therapeutic strategies, and future directionsYaw Ansong-Ansongton, Daisy Zapet Bamac, Angela Rivers
Journal of Comparative Psychology (Washington, D.C. : 1983)|November 19, 2008
Understanding of visual attention by adult humans (Homo sapiens): a partial replication of Povinelli, Bierschwale, and Cech (1999)Emily Thomas, Mary Murphy, Rebecca Pitt, et al.
Experimental Hematology|October 4, 2025
Mitochondrial retention and autophagy dysregulation drive oxidative stress in sickle cell disease erythrocytesJagadeesh Ramasamy, Prasanth Kumar Punathil Kannan, Sugasini Dhavamani, et al.
Epigenetics|May 2, 2015
Hydroxymethylcytosine and demethylation of the γ-globin gene promoter during erythroid differentiationMaria Armila Ruiz, Angela Rivers, Vinzon Ibanez, et al.
Ejhaem|August 28, 2025
Dual Assessment of the RBC Fraction Abnormally Retaining Mitochondria and of the RBC Mitochondrial Load Provides a New Clinical Parameter for Sickle Cell Disease PatientsEric Soupene, Hart Horneman, Mikail Alejandro, et al.
Journal of Pediatric Hematology/Oncology|June 27, 2024
Hereditary Spherocytosis with Mitochondrial Retention, Increased Oxidative Stress, and Alterations to Bioactive Membrane LipidsDaniel R Principe, Paige Reilly, Sugasini Dhavamani, et al.
Experimental Hematology|May 2, 2015
RN-1, a potent and selective lysine-specific demethylase 1 inhibitor, increases γ-globin expression, F reticulocytes, and F cells in a sickle cell disease mouse modelAngela Rivers, Kestis Vaitkus, Maria Armila Ruiz, et al.
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