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Science Advances|July 3, 2018
Podocyte injury elicits loss and recovery of cellular forcesKathryn E Haley, Nils M Kronenberg, Philipp Liehm, et al.Journal of Immunology (Baltimore, Md. : 1950)|September 7, 2001
Human NKT cells mediate antitumor cytotoxicity directly by recognizing target cell CD1d with bound ligand or indirectly by producing IL-2 to activate NK cellsL S Metelitsa, O V Naidenko, A Kant, et al.Development (Cambridge, England)|January 20, 2006
Nkx3.2/Bapx1 acts as a negative regulator of chondrocyte maturationSylvain Provot, Hervé Kempf, L Charles Murtaugh, et al.Endocrinology|November 1, 1996
Parathyroid hormone-related peptide delays terminal differentiation of chondrocytes during endochondral bone developmentK Lee, B Lanske, A C Karaplis, et al.Annals of the New York Academy of Sciences|February 13, 1996
Antigen-presenting function of the mouse CD1 moleculeS Tangri, H R Holcombe, A R Castaño, et al.Toxicology and Applied Pharmacology|January 1, 1997
Mechanism of thiazopyr-induced effects on thyroid hormone homeostasis in male Sprague-Dawley ratsK J Hotz, A G Wilson, D C Thake, et al.Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|March 27, 2025
Single cell RNA sequencing shows that cells expressing Sox9 postnatally populate most skeletal lineages in mouse boneNeal P Smith, Christopher Janton, Ana Clara Morellato Alcantara, et al.The Journal of Biological Chemistry|March 24, 1995
Parathyroid hormone (PTH)-PTH-related peptide hybrid peptides reveal functional interactions between the 1-14 and 15-34 domains of the ligandT J Gardella, M D Luck, A K Wilson, et al.Journal of Bone and Mineral Research : the Official Journal of the American Society for Bone and Mineral Research|August 8, 2008
Disruption of BMP signaling in osteoblasts through type IA receptor (BMPRIA) increases bone massNobuhiro Kamiya, Ling Ye, Tatsuya Kobayashi, et al.Science (New York, N.Y.)|July 14, 1995
Peptide binding and presentation by mouse CD1A R Castaño, S Tangri, J E Miller, et al.Pageof 39