B H Lorell|C S Apstein|E O Weinberg|M J Cunningham - JoVE Visualize

Showing results (1-10 of 265) with videos related to

Pageof 27

Sort By:

The American Journal of Physiology|March 1, 1989

Deleterious effect of ouabain on myocardial function during hypoxiaM J Cunningham, C S Apstein, E O Weinberg, et al.

European Heart Journal|November 1, 1990

Diastolic function in left ventricular hypertrophy: clinical and experimental relationshipsB H Lorell, C S Apstein, E O Weinberg, et al.

Circulation Research|February 1, 1990

Influence of glucose and insulin on the exaggerated diastolic and systolic dysfunction of hypertrophied rat hearts during hypoxiaM J Cunningham, C S Apstein, E O Weinberg, et al.

Circulation Research|August 1, 1988

Effects of ouabain and isoproterenol on left ventricular diastolic function during low-flow ischemia in isolated, blood-perfused rabbit heartsB H Lorell, S Isoyama, W N Grice, et al.

Circulation Research|August 1, 1990

Contribution of endothelial cells to calcium-dependent fluorescence transients in rabbit hearts loaded with indo 1B H Lorell, C S Apstein, M J Cunningham, et al.

Journal of Cardiac Surgery|December 1, 1988

The physiological basis of left ventricular diastolic dysfunctionC S Apstein, B H Lorell

The Journal of Physiology|October 15, 1994

Effects of angiotensin II on intracellular Ca2+ and pH in isolated beating rabbit hearts and myocytes loaded with the indicator indo-1H Ikenouchi, W H Barry, J H Bridge, et al.

Circulation Research|April 1, 1988

Enhanced sensitivity to hypoxia-induced diastolic dysfunction in pressure-overload left ventricular hypertrophy in the rat: role of high-energy phosphate depletionL F Wexler, B H Lorell, S Momomura, et al.

Hypertension (Dallas, Tex. : 1979)|April 1, 1989

Influence of hypertension with minimal hypertrophy on diastolic function during demand ischemiaB H Lorell, W N Grice, C S Apstein

Circulation Research|May 1, 1992

Exacerbation of left ventricular ischemic diastolic dysfunction by pressure-overload hypertrophy. Modification by specific inhibition of cardiac angiotensin converting enzymeF R Eberli, C S Apstein, S Ngoy, et al.

Pageof 27

Showing results (1-10 of 265) with videos related to

Sort By:

Pageof 27

The American Journal of Physiology|March 1, 1989

Deleterious effect of ouabain on myocardial function during hypoxiaM J Cunningham, C S Apstein, E O Weinberg, et al.

European Heart Journal|November 1, 1990

Diastolic function in left ventricular hypertrophy: clinical and experimental relationshipsB H Lorell, C S Apstein, E O Weinberg, et al.

Circulation Research|February 1, 1990

Influence of glucose and insulin on the exaggerated diastolic and systolic dysfunction of hypertrophied rat hearts during hypoxiaM J Cunningham, C S Apstein, E O Weinberg, et al.

Circulation Research|August 1, 1988

Effects of ouabain and isoproterenol on left ventricular diastolic function during low-flow ischemia in isolated, blood-perfused rabbit heartsB H Lorell, S Isoyama, W N Grice, et al.

Circulation Research|August 1, 1990

Contribution of endothelial cells to calcium-dependent fluorescence transients in rabbit hearts loaded with indo 1B H Lorell, C S Apstein, M J Cunningham, et al.

Journal of Cardiac Surgery|December 1, 1988

The physiological basis of left ventricular diastolic dysfunctionC S Apstein, B H Lorell

The Journal of Physiology|October 15, 1994

Effects of angiotensin II on intracellular Ca2+ and pH in isolated beating rabbit hearts and myocytes loaded with the indicator indo-1H Ikenouchi, W H Barry, J H Bridge, et al.

Circulation Research|April 1, 1988

Enhanced sensitivity to hypoxia-induced diastolic dysfunction in pressure-overload left ventricular hypertrophy in the rat: role of high-energy phosphate depletionL F Wexler, B H Lorell, S Momomura, et al.

Hypertension (Dallas, Tex. : 1979)|April 1, 1989

Influence of hypertension with minimal hypertrophy on diastolic function during demand ischemiaB H Lorell, W N Grice, C S Apstein

Circulation Research|May 1, 1992

Exacerbation of left ventricular ischemic diastolic dysfunction by pressure-overload hypertrophy. Modification by specific inhibition of cardiac angiotensin converting enzymeF R Eberli, C S Apstein, S Ngoy, et al.

Pageof 27