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Oncogene|June 2, 2018
Control of PD-L1 expression by miR-140/142/340/383 and oncogenic activation of the OCT4-miR-18a pathway in cervical cancerPeixin Dong, Ying Xiong, Jiehai Yu, et al.Vaccine|September 17, 2025
Predicting influenza vaccine-elicited antibody responses with practical point systemsYe Shen, Meng-Hsuan Sung, Yang Ge, et al.Journal of Experimental & Clinical Cancer Research : CR|September 7, 2016
MiR-137 and miR-34a directly target Snail and inhibit EMT, invasion and sphere-forming ability of ovarian cancer cellsPeixin Dong, Ying Xiong, Hidemichi Watari, et al.Journal of Inherited Metabolic Disease|October 27, 2004
Maternal Phenylketonuria Collaborative Study (MPKUCS)--the 'outliers'W B Hanley, C Azen, R Koch, et al.Vaccines|July 10, 2020
Japan's Ongoing Crisis on HPV VaccinationMasayuki Sekine, Risa Kudo, Manako Yamaguchi, et al.Vaccines|August 6, 2020
Epidemiologic Profile of Type-Specific Human Papillomavirus Infection after Initiation of HPV VaccinationMasayuki Sekine, Manako Yamaguchi, Risa Kudo, et al.Molecular Genetics and Metabolism|June 28, 2000
"Hypotyrosinemia" in phenylketonuriaW B Hanley, A W Lee, A J Hanley, et al.International Journal of Cancer|June 26, 2012
Incidence risk of cervical intraepithelial neoplasia 3 or more severe lesions is a function of human papillomavirus genotypes and severity of cytological and histological abnormalities in adult Japanese womenMasayoshi Hosaka, Hiromasa Fujita, Sharon J B Hanley, et al.American Journal of Obstetrics and Gynecology|April 1, 1992
Maternal phenylketonuria collaborative study, obstetric aspects and outcome: the first 6 yearsL D Platt, R Koch, C Azen, et al.The Journal of Clinical Investigation|March 11, 2000
Factors influencing T-cell turnover in HIV-1-seropositive patientsJ M McCune, M B Hanley, D Cesar, et al.Pageof 14