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Proceedings of the National Academy of Sciences of the United States of America|November 1, 1992
A mechanism for the antiinflammatory effects of corticosteroids: the glucocorticoid receptor regulates leukocyte adhesion to endothelial cells and expression of endothelial-leukocyte adhesion molecule 1 and intercellular adhesion molecule 1B N Cronstein, S C Kimmel, R I Levin, et al.Inflammation|June 1, 1994
Nonsteroidal antiinflammatory agents inhibit stimulated neutrophil adhesion to endothelium: adenosine dependent and independent mechanismsB N Cronstein, M Van de Stouwe, L Druska, et al.The Journal of Clinical Investigation|August 1, 1995
Colchicine alters the quantitative and qualitative display of selectins on endothelial cells and neutrophilsB N Cronstein, Y Molad, J Reibman, et al.Journal of Immunology (Baltimore, Md. : 1950)|April 1, 1992
Neutrophil adherence to endothelium is enhanced via adenosine A1 receptors and inhibited via adenosine A2 receptorsB N Cronstein, R I Levin, M Philips, et al.The Biochemical Journal|June 15, 1988
Occupancy of adenosine receptors raises cyclic AMP alone and in synergy with occupancy of chemoattractant receptors and inhibits membrane depolarizationB N Cronstein, S B Kramer, E D Rosenstein, et al.Journal of Immunology (Baltimore, Md. : 1950)|August 1, 1993
Chemoattraction of neutrophils by substance P and transforming growth factor-beta 1 is inadequately explained by current models of lipid remodelingK A Haines, S L Kolasinski, B N Cronstein, et al.Rheumatic Diseases Clinics of North America|November 15, 1997
The mechanism of action of methotrexateB N CronsteinJournal of Applied Physiology (Bethesda, Md. : 1985)|January 1, 1994
Adenosine, an endogenous anti-inflammatory agentB N CronsteinCurrent Opinion in Rheumatology|May 1, 1994
Adhesion molecules in the pathogenesis of rheumatoid arthritisB N CronsteinBritish Journal of Rheumatology|November 1, 1995
The antirheumatic agents sulphasalazine and methotrexate share an anti-inflammatory mechanismB N CronsteinPageof 20