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RSC Advances|May 2, 2022
The effects of microstructure, Nb content and secondary Ruddlesden-Popper phase on thermoelectric properties in perovskite CaMn1- Nb O3 (x = 0-0.10) thin filmsE Ekström, A le Febvrier, F Bourgeois, et al.
The Journal of Neuroscience : the Official Journal of the Society for Neuroscience|February 12, 2010
Complement component c1q mediates mitochondria-driven oxidative stress in neonatal hypoxic-ischemic brain injuryVadim S Ten, Jun Yao, Veniamin Ratner, et al.
Klinische Padiatrie|November 13, 2012
[Interdisciplinary AWMF guideline for the treatment of primary antibody deficiencies]J Krudewig, U Baumann, H Bernuth von, et al.
The Journal of Clinical Investigation|September 21, 2010
Human C3 mutation reveals a mechanism of dense deposit disease pathogenesis and provides insights into complement activation and regulationRubén Martínez-Barricarte, Meike Heurich, Francisco Valdes-Cañedo, et al.
American Journal of Physiology. Renal Physiology|March 17, 2023
Tacrolimus induces fibroblast-to-myofibroblast transition via a TGF-β-dependent mechanism to contribute to renal fibrosisAdaku C Ume, Tara Y Wenegieme, Jennae N Shelby, et al.
American Journal of Human Genetics|September 26, 2002
Autosomal dominant familial calcium pyrophosphate dihydrate deposition disease is caused by mutation in the transmembrane protein ANKHCharlene J Williams, Yun Zhang, Andrew Timms, et al.
Bone|January 2, 2016
Deletion of the membrane complement inhibitor CD59a drives age and gender-dependent alterations to bone phenotype in miceAnja C Bloom, Fraser L Collins, Rob J Van't Hof, et al.
Molecular Immunology|January 31, 2016
Genetic analysis and functional characterization of novel mutations in a series of patients with atypical hemolytic uremic syndromeNóra Szarvas, Ágnes Szilágyi, Dorottya Csuka, et al.
The Journal of Infectious Diseases|December 31, 2021
Efficacy of an Experimental Gonococcal Lipooligosaccharide Mimitope Vaccine Requires Terminal ComplementLisa A Lewis, Sunita Gulati, Wioleta M Zelek, et al.
Blood|February 19, 2008
Circulating neutrophils maintain physiological blood pressure by suppressing bacteria and IFNgamma-dependent iNOS expression in the vasculature of healthy miceJonathan Morton, Barbara Coles, Kate Wright, et al.
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