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Clinical Chemistry|May 1, 1989
An automated procedure for measuring biotinidase activity in serumK A Weissbecker, H D Gruemer, G S Heard, et al.Proceedings of the National Academy of Sciences of the United States of America|July 20, 2022
Developmental constraints enforce altruism and avert the tragedy of the commons in a social microbeLaurence J Belcher, Philip G Madgwick, Satoshi Kuwana, et al.Journal of Inherited Metabolic Disease|May 9, 2000
Novel mutations cause biotinidase deficiency in Turkish childrenR J Pomponio, T Coskun, M Demirkol, et al.Seminars in Neurology|January 23, 2023
Challenging Cases in NeuroimmunologyAnna A Shah, Andrew B Wolf, Anthony Declusin, et al.Journal of Neurophysiology|July 24, 2015
An integrative role for the superior colliculus in selecting targets for movementsAndrew B Wolf, Mario J Lintz, Jamie D Costabile, et al.Plos Genetics|September 21, 2011
Genetic effects at pleiotropic loci are context-dependent with consequences for the maintenance of genetic variation in populationsHeather A Lawson, Janet E Cady, Charlyn Partridge, et al.Human Genetics|July 8, 1998
Partial biotinidase deficiency is usually due to the D444H mutation in the biotinidase geneK L Swango, M Demirkol, G Hüner, et al.Human Mutation|April 17, 1999
Double mutation (A171T and D444H) is a common cause of profound biotinidase deficiency in children ascertained by newborn screening the the United States. Mutations in brief no. 128. OnlineK J Norrgard, R J Pomponio, K L Swango, et al.Archives of Orthopaedic and Trauma Surgery|September 17, 2008
Ulnar shortening after TFCC suture repair of Palmer type 1B lesionsMaya B Wolf, Markus W Kroeber, Andreas Reiter, et al.Current Biology : CB|March 31, 2015
Fitness Trade-offs Result in the Illusion of Social SuccessJason B Wolf, Jennifer A Howie, Katie Parkinson, et al.Pageof 69