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American Journal of Hypertension|September 12, 2009
Effects of 17-hydroxyprogesterone on tumor necrosis factor-alpha-induced hypertension during pregnancySharon D Keiser, Edward W Veillon, Marc R Parrish, et al.
Journal of Hypertension : Open Access|November 18, 2014
CD4+ T Cells Play a Critical Role in Mediating Hypertension in Response to Placental IschemiaSarah Novotny, Kedra Wallace, Florian Herse, et al.
American Journal of Obstetrics and Gynecology|February 20, 2014
Progesterone supplementation attenuates hypertension and the autoantibody to the angiotensin II type I receptor in response to elevated interleukin-6 during pregnancyLorena M Amaral, Luissa Kiprono, Denise C Cornelius, et al.
American Journal of Physiology. Regulatory, Integrative and Comparative Physiology|October 28, 2016
Reduced uterine perfusion pressure T-helper 17 cells cause pathophysiology associated with preeclampsia during pregnancyDenise C Cornelius, Lorena M Amaral, Kedra Wallace, et al.
American Journal of Physiology. Regulatory, Integrative and Comparative Physiology|February 28, 2019
Natural killer cells contribute to mitochondrial dysfunction in response to placental ischemia in reduced uterine perfusion pressure ratsVenkata Ramana Vaka, Kristen M McMaster, Denise C Cornelius, et al.
Hypertension (Dallas, Tex. : 1979)|July 18, 2018
Role of Mitochondrial Dysfunction and Reactive Oxygen Species in Mediating Hypertension in the Reduced Uterine Perfusion Pressure Rat Model of PreeclampsiaVenkata Ramana Vaka, Kristen M McMaster, Mark W Cunningham, et al.
American Journal of Physiology. Renal Physiology|November 16, 2020
CD4+ T cells cause renal and placental mitochondrial oxidative stress as mechanisms of hypertension in response to placental ischemiaEvangeline Deer, Kristin E Reeve, Lorena Amaral, et al.
Hypertension (Dallas, Tex. : 1979)|March 21, 2018
AT1-AA (Angiotensin II Type 1 Receptor Agonistic Autoantibody) Blockade Prevents Preeclamptic Symptoms in Placental Ischemic RatsMark W Cunningham, Javier Castillo, Tarek Ibrahim, et al.
Journal of the American Heart Association|February 14, 2024
AT1-AA Is Produced in Offspring in Response to Placental Ischemia and Is Lowered by B-Cell Depletion Without Compromising Overall Offspring HealthNathan Campbell, Evangeline Deer, Dylan Solise, et al.
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