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Journal of Cardiovascular Translational Research|July 15, 2010
Development of hydrogen sulfide-based therapeutics for cardiovascular diseaseBenjamin L Predmore, David J Lefer
Expert Review of Clinical Pharmacology|March 5, 2011
Hydrogen sulfide-mediated myocardial pre- and post-conditioningBenjamin L Predmore, David J Lefer
Frontiers in Physiology|December 24, 2011
Hydrogen sulfide increases nitric oxide production from endothelial cells by an akt-dependent mechanismBenjamin L Predmore, David Julian, Arturo J Cardounel
Age (Dordrecht, Netherlands)|May 27, 2010
The hydrogen sulfide signaling system: changes during aging and the benefits of caloric restrictionBenjamin L Predmore, Maikel J Alendy, Khadija I Ahmed, et al.
Physiological and Biochemical Zoology : PBZ|March 31, 2009
Hydrogen sulfide induces oxidative damage to RNA and DNA in a sulfide-tolerant marine invertebrateJoanna Joyner-Matos, Benjamin L Predmore, Jenny R Stein, et al.
Diabetes Technology & Therapeutics|April 4, 2012
Single-dose pharmacokinetics of different oral sodium nitrite formulations in diabetes patientsFrank L Greenway, Benjamin L Predmore, Douglas R Flanagan, et al.
Transfusion|March 14, 2013
Red blood cells stored for increasing periods produce progressive impairments in nitric oxide-mediated vasodilationJason T Alexander, Alexander M El-Ali, James L Newman, et al.
Arteriosclerosis, Thrombosis, and Vascular Biology|June 2, 2012
Selective β2-adrenoreceptor stimulation attenuates myocardial cell death and preserves cardiac function after ischemia-reperfusion injuryShashi Bhushan, Kazuhisa Kondo, Benjamin L Predmore, et al.
American Journal of Physiology. Heart and Circulatory Physiology|April 3, 2012
The polysulfide diallyl trisulfide protects the ischemic myocardium by preservation of endogenous hydrogen sulfide and increasing nitric oxide bioavailabilityBenjamin L Predmore, Kazuhisa Kondo, Shashi Bhushan, et al.
Journal of the American College of Cardiology|December 14, 2011
Beta3-adrenoreceptor stimulation ameliorates myocardial ischemia-reperfusion injury via endothelial nitric oxide synthase and neuronal nitric oxide synthase activationJuan P Aragón, Marah E Condit, Shashi Bhushan, et al.
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